Article(id=1319291469500891581, tenantId=1146029695717560320, journalId=1255847919539208197, issueId=1319291312826863771, articleNumber=null, orderNo=null, doi=10.13193/j.issn.1673-7717.2026.08.042, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=null, receivedDateStr=null, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1792473197579, onlineDateStr=2026-10-20, pubDate=1786291200000, pubDateStr=2026-08-10, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1792473197579, onlineIssueDateStr=2026-10-20, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1792473197579, creator=13701087609, updateTime=1792473197579, updator=13701087609, issue=Issue{id=1319291312826863771, tenantId=1146029695717560320, journalId=1255847919539208197, year='2026', volume='44', issue='8', pageStart='1', pageEnd='258', issueExtLink='null', onlineDate='null', pubDate='1786291200000', pubDateStr='2026-08-10', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=null, createTime=1792473160225, creator='13701087609', updateTime=1792473382588, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1319292245547799195, tenantId=1146029695717560320, journalId=1255847919539208197, issueId=1319291312826863771, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1319292245652656796, tenantId=1146029695717560320, journalId=1255847919539208197, issueId=1319291312826863771, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=208, endPage=211, ext={EN=ArticleExt(id=1319291469731578302, articleId=1319291469500891581, tenantId=1146029695717560320, journalId=1255847919539208197, language=EN, title=Effects of Modified Sijunzi Decoction (四君子汤加减) on 5-HT Receptor, Serum Inflammation and Brain-Gut Peptide in Patients with Irritable bowel syndrome, columnId=1256183365644968140, journalTitle=Chinese Archives of Traditional Chinese Medicine, columnName=Platform of Provincial Project, runingTitle=null, highlight=null, articleAbstract=
Objective To explore the effects of Modified Sijunzi Decoction (四君子汤加减) on 5-hydroxytryptamine (5-HT) receptor, serum inflammation and brain-gut peptide in patients with irritable bowel syndrome (IBS).
Methods A total of 122 IBS patients were randomly divided into Sijunzi Decoction group and control group (61 cases in each group). Both groups were treated with western medicine, and the Sijunzi Decoction group was treated with Modified Sijunzi Decoction on the basis of western medicine and the treatment time was 28 d. The changes of TCM syndrome integral, 5-HT receptor mRNA of intestinal mucosa, serum inflammation and brain-gut peptide were observed.
Results There was no statistically significant difference in traditional Chinese medicine syndrome scores between the two groups before treatment (P>0.05). After treatment, both groups had a decrease in scores (P<0.05), and the score of the Sijunzi Decoction group was lower than that of the control group (P<0.05). There was no statistically significant difference in the expression levels of 5-HT receptors between the two groups before treatment (P>0.05). After a period of treatment, both groups showed a decrease (P<0.05), and level of the Sijunzi Decoction group was lower than that of the control group (P<0.05). There was no statistically significant difference in serum inflammatory cytokine levels between the two groups before treatment (P>0.05). The serum levels of interleukin (IL)-8, IL-10, IL-12 and tumor necrosis factor-α (TNF-α) all decreased after treatment (P<0.05), and the levels of the Sijunzi Decoction group were lower than those of the control group (P<0.05), while IL-10 was higher than before treatment (P<0.05), and the level of the Sijunzi Decoction group was higher than that of the control group (P<0.05). There was no statistically significant difference in brain gut peptide levels between the two groups before treatment (P>0.05). After treatment, the levels of vasoactine intrestinal peptide (VIP), calcitonin gene-related peptide (CGRP) and 5-HT in both groups decreased compared to those before treatment (P<0.05), and the levels of the Sijunzi Decoction group was lower than those of the control group (P<0.05). Neuropeptide Y (NPY) levels all increased (P<0.05), and the level of the Sijunzi Decoction group was higher than that of the control group (P<0.05). The total effective rates of the Sijunzi Decoction group and the control group were 91.80% (56/61) and 75.41% (46/61), respectively, with statistical differences (P<0.05) between the two groups.
CONCLUSION Sijunzi Decoction can significantly improve the clinical symptoms and improve the efficacy of IBS. The mechanism may be related to regulating the expression of 5-HT receptor in intestinal mucosa, improving the level of brain-intestinal peptide, and inhibiting inflammatory response.
, authors=Wen WU, Junping WANG, Jiandong ZHAO, Yourui ZHANG, Haosen HAO, Yu QIAO, authorsList=Wen WU, Junping WANG, Jiandong ZHAO, Yourui ZHANG, Haosen HAO, Yu QIAO, authorCompany=null, correspAuthors=Jiandong ZHAO, authorNote=null, correspAuthorsNote=null, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1319291470188757439, articleId=1319291469500891581, tenantId=1146029695717560320, journalId=1255847919539208197, language=CN, title=四君子汤加减对肠易激综合征患者5-羟色胺受体、血清炎症因子和脑肠肽的影响研究, columnId=1256183369033965799, journalTitle=中华中医药学刊, columnName=省级项目平台, runingTitle=null, highlight=null, articleAbstract=
目的 探讨四君子汤加减对肠易激综合征（irritable bowel syndrome，IBS）患者5-羟色胺（5-hydroxytryptamine，5-HT）受体、血清炎症和脑肠肽的影响。
方法 选取IBS患者122例，随机分为两组，每组61例，对照组常规给予西药治疗，四君子汤组在对照组基础上给予四君子汤加减联合治疗，疗程28 d，观察治疗前后中医证候积分、肠黏膜5-HT受体mRNA、血清炎症因子及脑肠肽水平变化。
结果 两组患者治疗前中医证候积分比较差异无统计学意义（P>0.05），治疗后，两组积分均有所下降（P<0.05），且四君子汤组低于对照组（P<0.05）。两组患者治疗前5-HT受体表达水平比较差异无统计学意义（P>0.05），经过一段时间治疗后两组均有所下降（P<0.05），且四君子汤组低于对照组（P<0.05）。两组患者在治疗前血清炎症因子水平比较差异无统计学意义（P>0.05），血清白细胞介素（interleukin，IL）-8、IL-10、IL-12、肿瘤坏死因子-α（tumor necrosis factor-α，TNF-α）水平治疗后均有所下降（P<0.05），且四君子汤组低于对照组（P<0.05），IL-10较治疗前均升高（P<0.05），且四君子汤组高于对照组（P<0.05）。两组患者治疗前脑肠肽水平比较差异无统计学意义（P>0.05），治疗后两组血管活性肠肽（vasoactine intrestinal peptide，VIP）、降钙素基因相关肽（calcitonin gene-related peptide，CGRP）、5-HT水平均较治疗前下降（P<0.05），且四君子汤组低于对照组（P<0.05）；神经肽Y（neuropeptide Y，NPY）均升高（P<0.05），且四君子汤组高于对照组（P<0.05）。四君子汤组和对照组的治疗总有效率分别为91.80%（56/61）和75.41%（46/61），两组比较差异具有统计学意义（P<0.05）。
结论 四君子汤加减用于治疗IBS能显著改善患者临床症状，提高治疗疗效，其机制可能与调节肠黏膜5-HT受体表达进而起到改善脑肠肽水平、抑制炎症反应等有关。
, authors=武雯, 王俊平, 赵建栋, 张佑蕊, 郝浩森, 乔钰, authorsList=武雯, 王俊平, 赵建栋, 张佑蕊, 郝浩森, 乔钰, authorCompany=null, correspAuthors=赵建栋, authorNote=
武雯（1984-），女，山西太原人，主治医师，硕士，研究方向：中西医结合诊治消化系统疾病。
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武雯（1984-），女，山西太原人，主治医师，硕士，研究方向：中西医结合诊治消化系统疾病。
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武雯（1984-），女，山西太原人，主治医师，硕士，研究方向：中西医结合诊治消化系统疾病。
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