Article(id=1319291438043615405, tenantId=1146029695717560320, journalId=1255847919539208197, issueId=1319291312826863771, articleNumber=null, orderNo=null, doi=10.13193/j.issn.1673-7717.2026.08.051, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=null, receivedDateStr=null, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1792473190079, onlineDateStr=2026-10-20, pubDate=1786291200000, pubDateStr=2026-08-10, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1792473190079, onlineIssueDateStr=2026-10-20, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1792473190079, creator=13701087609, updateTime=1792473190079, updator=13701087609, issue=Issue{id=1319291312826863771, tenantId=1146029695717560320, journalId=1255847919539208197, year='2026', volume='44', issue='8', pageStart='1', pageEnd='258', issueExtLink='null', onlineDate='null', pubDate='1786291200000', pubDateStr='2026-08-10', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=null, createTime=1792473160225, creator='13701087609', updateTime=1792473382588, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1319292245547799195, tenantId=1146029695717560320, journalId=1255847919539208197, issueId=1319291312826863771, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1319292245652656796, tenantId=1146029695717560320, journalId=1255847919539208197, issueId=1319291312826863771, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=246, endPage=249, ext={EN=ArticleExt(id=1319291438282690734, articleId=1319291438043615405, tenantId=1146029695717560320, journalId=1255847919539208197, language=EN, title=Exploring Innovative Pathogenesis and Prevention-Treatment Models for Heart Failure from erspective of Yin and Yang Linking Vessels, columnId=1256183365644968140, journalTitle=Chinese Archives of Traditional Chinese Medicine, columnName=Platform of Provincial Project, runingTitle=null, highlight=null, articleAbstract=
It explored the pathogenesis and therapeutic strategies for chronic heart failure (CHF) based on the theory of “Yin and Yang Link Vessels”. In Traditional Chinese Medicine (TCM), the key pathogenesis of CHF involves an imbalance of Qi, blood, Yin and Yang, leading to a pathological pattern of root deficiency and excess manifestations characterized by the intermingling of deficiency, blood stasis and fluid retention. Building upon existing CHF theories (e.g., Qi deficiency with blood stasis, Yang deficiency with water overflow and heart-kidney Yang deficiency), this study further proposed that the disharmony of the Yin and Yang Link Vessels was a critical pathogenic mechanism. Accordingly, a “harmonizing the link vessels and unblocking collaterals” therapeutic model was established, employing a comprehensive treatment approach that included boosting Qi and warming Yang, activating blood circulation and unblocking collaterals, and resolving fluid retention and promoting diuresis, with an emphasis on individualized and dynamic pattern differentiation. This theory provides novel insights for TCM diagnosis and treatment of CHF, expands the application of TCM theories in cardiovascular diseases, and holds significant clinical value.
, authors=Sitong YAN
1, Ying YANG
2, Jinxia WANG
2, authorsList=Sitong YAN, Ying YANG, Jinxia WANG, authorCompany=null, correspAuthors=Ying YANG, authorNote=null, correspAuthorsNote=null, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1319291438651789487, articleId=1319291438043615405, tenantId=1146029695717560320, journalId=1255847919539208197, language=CN, title=阴阳维脉视角下心力衰竭创新病机及防治模式探讨, columnId=1256183369033965799, journalTitle=中华中医药学刊, columnName=省级项目平台, runingTitle=null, highlight=null, articleAbstract=
基于“阴阳维脉”理论,探讨慢性心力衰竭的发病机制及防治思路。心力衰竭的中医病机主要为气血阴阳失衡,从而形成虚、瘀、水互结的本虚标实病理格局。在现有心力衰竭理论(气虚血瘀、阳虚水泛、心肾阳虚等)基础上,补充提出阴阳维脉失和是心力衰竭发生的关键病机,并据此构建“和维通络”防治模式,采用益气温阳、活血通络、化饮利水的综合治法,注重个体化与动态辨治。为心力衰竭中医诊疗提供新思路,并拓展了中医理论在心血管领域的应用,具有重要临床价值。
, authors=闫思彤
1, 杨莺
2, 王金侠
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闫思彤(2000-),女,辽宁海城人,硕士在读,研究方向:中西医结合防治心血管疾病。
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