Article(id=1317115133952906237, tenantId=1146029695717560320, journalId=1255847919539208197, issueId=1317114999827452826, articleNumber=null, orderNo=null, doi=10.13193/j.issn.1673-7717.2026.06.037, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=null, receivedDateStr=null, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1791954318749, onlineDateStr=2026-10-14, pubDate=1781020800000, pubDateStr=2026-06-10, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1791954318749, onlineIssueDateStr=2026-10-14, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1791954318749, creator=13701087609, updateTime=1791954318749, updator=13701087609, issue=Issue{id=1317114999827452826, tenantId=1146029695717560320, journalId=1255847919539208197, year='2026', volume='44', issue='6', pageStart='1', pageEnd='258', issueExtLink='null', onlineDate='null', pubDate='1781020800000', pubDateStr='2026-06-10', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=null, createTime=1791954286772, creator='13701087609', updateTime=1791958677365, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1317133415753831385, tenantId=1146029695717560320, journalId=1255847919539208197, issueId=1317114999827452826, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1317133415753831386, tenantId=1146029695717560320, journalId=1255847919539208197, issueId=1317114999827452826, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=199, endPage=202, ext={EN=ArticleExt(id=1317115134594634750, articleId=1317115133952906237, tenantId=1146029695717560320, journalId=1255847919539208197, language=EN, title=Exploring Effect of Ginkgo Biloba Extract on Inflammatory Response in Rats with Ulcerative Colitis Based on TLR4/NF-κB Signaling Pathway, columnId=1256183361521967297, journalTitle=Chinese Archives of Traditional Chinese Medicine, columnName=Target on National Project, runingTitle=null, highlight=null, articleAbstract=
Objective To investigate the effects of Ginkgo biloba extract on the inflammatory response in the rats with ulcerative colitis (UC) based on the Toll-like receptor 4 (TLR4) /nuclear factor-κB (NF-κB) signaling pathway.
Methods Forty SD rats were divided model group, Ginkgo biloba extract low-dose group, Ginkgo biloba extract high-dose group and normal group, each with 10 rats. The levels of interleukin (IL) -1 β, IL-6 and tumor necrosis factor (TNF) -α were measured using the Enzyme linked immunosorbent assay (ELISA) method. The expressions of TLR4 and NF-κB proteins in colon tissue was assessed via Western blot (WB). The Disease Activity Index (DAI), colon length, colon mucosal damage index (CMDI), inflammatory factors, TLR4 and NF-κB protein expressions were compared among the groups.
Result The DAI score, colon mucosal damage index (CMDI) score, the protein gray values of IL-1β, IL-6, TNF-α, TLR4 and NF-κB in the model group, Ginkgo biloba extract low-dose group and Ginkgo biloba extract high-dose group were higher than those of the normal group, while the colon length was shorter than that of the normal group (P<0.05). The DAI score, CMDI score, the protein gray values of IL-1β, IL-6,TNF-α, TLR4 and NF-κB of the Ginkgo biloba extract low-dose and high-dose groups were lower than those of the model group, while the colon length was longer than that of the model group (P<0.05). The DAI score, CMDI score, the protein gray values of IL-1β, IL-6, TNF-α, TLR4 and NF-κB of the Ginkgo biloba extract high-dose group were lower than those of the Ginkgo biloba extract low-dose group, while the colon length was longer than that of the low-dose Ginkgo biloba extract group (P<0.05).
Conclusion Ginkgo biloba extract can alleviate the inflammatory response in the UC rats, and its mechanism may be related to the regulation of TLR4/NF-κB signaling pathway expression.
, authors=Zhili ZHENG
1, 2, Qiongqiong LU
3, Yuan DING
2, Wangyu YE
2, Han YU
4, authorsList=Zhili ZHENG, Qiongqiong LU, Yuan DING, Wangyu YE, Han YU, authorCompany=null, correspAuthors=Han YU, authorNote=null, correspAuthorsNote=null, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1317115134863070207, articleId=1317115133952906237, tenantId=1146029695717560320, journalId=1255847919539208197, language=CN, title=基于TLR4/NF-κB信号通路探讨银杏提取物对溃疡性结肠炎大鼠炎症反应影响, columnId=1256183362172142382, journalTitle=中华中医药学刊, columnName=国家项目点击, runingTitle=null, highlight=null, articleAbstract=
目的 探讨基于Toll样受体（Toll-like receptor 4，TLR4）/核转录因子-κB（nuclear factor-κB，NF-κB）信号通路探讨银杏提取物对溃疡性结肠炎（ulcerative colitis，UC）大鼠炎症反应影响。
方法 选取40只SD大鼠，随机分为模型组、银杏提取物低剂量组、银杏提取物高剂量组和正常组，各10只。采用酶联免疫吸附法（Enzyme linked immunosorbent assay，ELISA）法测定白介素（interleukin，IL）-1 β、IL-6和肿瘤坏死因子（tumor necrosis factor，TNF）-α水平；采用WB法测定结肠组织TLR4和NF-κB蛋白表达。比较各组大鼠UC疾病活动指数（Disease Activity Index，DAI），结肠长度，结肠黏膜损伤指数（colon mucosal damage index，CMDI），炎症因子，TLR4和NF-κB蛋白表达。
结果 模型组、银杏提取物低剂量组和银杏提取物高剂量组DAI评分、CMDI评分、IL-1β、IL-6、TNF-α、TLR4蛋白灰度值和NF-κB蛋白灰度值高于正常组，而结肠长度短于正常组（P<0.05）；银杏提取物低剂量组和银杏提取物高剂量组DAI评分、CMDI评分、IL-1β、IL-6、TNF-α、TLR4蛋白灰度值和NF-κB蛋白灰度值低于模型组，而结肠长度长于模型组（P<0.05）；银杏提取物高剂量组DAI评分、CMDI评分、IL-1β、IL-6、TNF-α、TLR4蛋白灰度值和NF-κB蛋白灰度值低于银杏提取物低剂量组，而结肠长度长于银杏提取物低剂量组（P<0.05）。
结论 银杏提取物可减轻UC大鼠炎症反应，其机制可能与调控TLR4/NF-κB信号通路表达有关。
, authors=郑智礼
1, 2, 路琼琼
3, 丁媛
2, 叶王宇
2, 于瀚
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郑智礼（1992-），女，河南焦作人，主治医师，博士，研究方向：中医药防治消化系统疾病。
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