Article(id=1317115124498936750, tenantId=1146029695717560320, journalId=1255847919539208197, issueId=1317115025307845060, articleNumber=null, orderNo=null, doi=10.13193/j.issn.1673-7717.2026.05.022, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=null, receivedDateStr=null, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1791954316495, onlineDateStr=2026-10-14, pubDate=1778342400000, pubDateStr=2026-05-10, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1791954316495, onlineIssueDateStr=2026-10-14, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1791954316495, creator=13701087609, updateTime=1791954316495, updator=13701087609, issue=Issue{id=1317115025307845060, tenantId=1146029695717560320, journalId=1255847919539208197, year='2026', volume='44', issue='5', pageStart='1', pageEnd='258', issueExtLink='null', onlineDate='null', pubDate='1778342400000', pubDateStr='2026-05-10', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=null, createTime=1791954292847, creator='13701087609', updateTime=1791958713604, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1317133567340171275, tenantId=1146029695717560320, journalId=1255847919539208197, issueId=1317115025307845060, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1317133567340171276, tenantId=1146029695717560320, journalId=1255847919539208197, issueId=1317115025307845060, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=115, endPage=119, ext={EN=ArticleExt(id=1317115124683486127, articleId=1317115124498936750, tenantId=1146029695717560320, journalId=1255847919539208197, language=EN, title=Exploration on Role of Macrophage Polarization in Coronary Microvascular Dysfunction and TCM Intervention Based On Theory of Healthy Qi and Pathogenic Factors, columnId=1256183361521967297, journalTitle=Chinese Archives of Traditional Chinese Medicine, columnName=Target on National Project, runingTitle=null, highlight=null, articleAbstract=

Macrophages acquire specific functional phenotypes secrete different cytokines and participate in the process of coronary microvascular dysfunction through polarization processes. The theory of healthy Qi and pathogenic factors in traditional Chinese medicine is an important guiding theory for clinical practice, closely related to the occurrence and development of coronary microvascular dysfunction. This article mainly discussed the role of macrophage polarization in coronary microvascular dysfunction, the relationship between the theory of healthy Qi and pathogenic factors and coronary microvascular dysfunction, the correlation between the theory of healthy Qi and pathogenic factors and macrophage polarization, and traditional Chinese medicine intervention. From the perspective of the theory of healthy Qi and pathogenic factors, it explored the role of macrophage polarization in coronary microvascular dysfunction, in order to explain the pathogenesis of coronary microvascular dysfunction and provide theoretical references for the clinical treatment of coronary microvascular dysfunction.

, authors=Yi LI¹, Yan ZHANG², Qirui MU¹, authorsList=Yi LI, Yan ZHANG, Qirui MU, authorCompany=null, correspAuthors=Yan ZHANG, authorNote=null, correspAuthorsNote=null, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1317115124901589936, articleId=1317115124498936750, tenantId=1146029695717560320, journalId=1255847919539208197, language=CN, title=基于正邪理论探讨巨噬细胞极化在冠脉微循环障碍中的作用及中药干预, columnId=1256183362172142382, journalTitle=中华中医药学刊, columnName=国家项目点击, runingTitle=null, highlight=null, articleAbstract=

巨噬细胞通过极化过程获得特定的功能表型，分泌不同的细胞因子，参与冠脉微循环障碍进程。中医正邪理论是指导临床的重要理论，与冠脉微循环障碍的发生发展密切相关。主要从巨噬细胞极化在冠脉微循环障碍中的作用、正邪理论与冠脉微循环障碍、正邪理论与巨噬细胞极化相关性及中药干预等方面进行论述，从正邪理论的角度探究巨噬细胞极化在冠脉微循环障碍中的作用，以期阐释冠脉微循环障碍的发病机制，为冠脉微循环障碍的临床治疗提供理论参考。

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李艺（1996-），女，辽宁沈阳人，博士在读，研究方向：中医药防治心脑血管疾病。

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李艺（1996-），女，辽宁沈阳人，博士在读，研究方向：中医药防治心脑血管疾病。

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