Article(id=1317115160293134577, tenantId=1146029695717560320, journalId=1255847919539208197, issueId=1317114999827452826, articleNumber=null, orderNo=null, doi=10.13193/j.issn.1673-7717.2026.06.047, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=null, receivedDateStr=null, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1791954325029, onlineDateStr=2026-10-14, pubDate=1781020800000, pubDateStr=2026-06-10, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1791954325029, onlineIssueDateStr=2026-10-14, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1791954325029, creator=13701087609, updateTime=1791954325029, updator=13701087609, issue=Issue{id=1317114999827452826, tenantId=1146029695717560320, journalId=1255847919539208197, year='2026', volume='44', issue='6', pageStart='1', pageEnd='258', issueExtLink='null', onlineDate='null', pubDate='1781020800000', pubDateStr='2026-06-10', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=null, createTime=1791954286772, creator='13701087609', updateTime=1791958677365, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1317133415753831385, tenantId=1146029695717560320, journalId=1255847919539208197, issueId=1317114999827452826, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1317133415753831386, tenantId=1146029695717560320, journalId=1255847919539208197, issueId=1317114999827452826, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=243, endPage=247, ext={EN=ArticleExt(id=1317115160515432690, articleId=1317115160293134577, tenantId=1146029695717560320, journalId=1255847919539208197, language=EN, title=Safety and Efficacy of Xuezhikang (血脂康) in Combination with PCSK9 Inhibitors in Treatment of Statin-Intolerant Patients with Coronary Atherosclerotic Heart Disease, columnId=1256183365644968140, journalTitle=Chinese Archives of Traditional Chinese Medicine, columnName=Platform of Provincial Project, runingTitle=null, highlight=null, articleAbstract=
Objective To observe the safety and efficacy of Xuezhikang (血脂康) in combination with proprotein convertase subtilisin/kexin type 9 (PCSK9i) in the treatment of the patients with statin-intolerant coronary atherosclerotic heart disease (coronary artery disease).
Methods A total of 104 cases of statin-intolerant coronary heart disease patients admitted to the Department of Cardiology of the Affiliated Hospital of Jilin Medical University from 2023.2 to 2024.2 were selected as the study population, and were divided into the routine group and the Xuezhikang group using the lottery method, and both groups were given coronary heart disease secondary preventive drug treatment, 52 cases in the routine group were given iloyuzumab treatment, 52 cases in the Xuezhikang group were given Xuezhikang Capsule combined with iloyuzumab treatment, and both groups were continuously treated for half a year. The lipid levels of the two groups before treatment, after 1, 3 and 6 months of treatment were compared. The levels of PCSK9, cholesterol ester transfer protein (CETP), apolipoprotein B (ApoB), cardiac function, 6-minute walk test, glutamic acid aminotransferase (ALT), glutamine transaminase (AST), creatine kinase isoenzymes (CK-MB) and traditional Chinese medicine syndrome scores in the two groups before treatment and after 6 months of treatment were compared. The cumulative endpoint events in both groups within six months were counted.
Results There was no difference in the levels of lipid indexes before treatment between the two groups (P>0.05). The levels of total cholesterol (TG), triglyceride (TC) and low-density lipoprotein cholesterol (LDL-C) after 1, 3 and 6 months of treatment in both groups were lower than those before treatment, and the levels of TG, TC and LDL-C in the Xuezhikang group were lower than those of the routine group after 1, 3 and 6 months of treatment (P<0.05). Comparison of high-density lipoprotein cholesterol (HDL-C) levels after 1, 3 and 6 months of treatment in the two groups showed no difference (P>0.05). Comparison of PCSK9, CETP, ApoB, AST, ALT, and CK-MB levels before treatment between the two groups showed no difference (P>0.05). The levels of PCSK9, ApoB, AST, ALT and CK-MB were lower than those before treatment after 6 months of treatment in both groups, and the levels of PCSK9 and ApoB in the Xuezhikang group were lower than those in the routine group after 6 months of treatment (P<0.05). There was no difference in the levels of CETP, AST, ALT or CK-MB after 6 months of treatment between the two groups (P>0.05). Comparison of cardiac function and 6-minute walk test levels before treatment between the two groups showed no difference (P>0.05). The levels of left ventricular ejection fraction (LVEF), cardiac output (CO) and 6-minute walk test were higher in both groups after 6 months of treatment compared with the pre-treatment levels, and the levels of LVEF, CO and 6-minute walk test were higher in the Xuezhikang group than those in the routine group after 6 months of treatment (P<0.05). The levels of left ventricular end-diastolic internal diameter (LVESD) were lower in both groups after 6 months of treatment than those before treatment, and the levels of LVESD were lower in the Xuezhikang group than those in the routine group after 6 months of treatment (P<0.05). Comparison of cumulative endpoint events between the two groups showed no difference (P>0.05). Before treatment, there was no difference in traditional Chinese medicine syndrome scores between the two groups (P>0.05). The scores of the two groups after 6 months of treatment were lower than those before treatment, and the score of the Xuezhikang group was lower than that of the routine group (P<0.05).
Conclusion Xuezhikang combined with PCSK9 inhibitor for statin-intolerant coronary artery disease can regulate the lipid metabolism and improve the cardiac function with high safety.
, authors=Jun WU
1, Xiaopeng LYU
2, Yueqing ZHANG
1, Yi LIU
3, authorsList=Jun WU, Xiaopeng LYU, Yueqing ZHANG, Yi LIU, authorCompany=null, correspAuthors=null, authorNote=null, correspAuthorsNote=null, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1317115160834199795, articleId=1317115160293134577, tenantId=1146029695717560320, journalId=1255847919539208197, language=CN, title=血脂康联合PCSK9抑制剂治疗他汀不耐受冠状动脉粥样硬化性心脏病的安全性和有效性, columnId=1256183369033965799, journalTitle=中华中医药学刊, columnName=省级项目平台, runingTitle=null, highlight=null, articleAbstract=
目的 观察血脂康联合前蛋白转化酶枯草溶菌素9抑制剂（proprotein convertase subtilisin/kexin type 9 inhibitor，PCSK9i）治疗他汀不耐受冠状动脉粥样硬化性心脏病（简称冠心病）患者的安全性和有效性。
方法 选取吉林医药学院附属医院心内科2023年2月—2024年2月收治的104例他汀不耐受冠心病患者为研究人群，采用抽签法分为常规组和血脂康组，两组均给予冠心病二级预防药物治疗，常规组52例给予依洛尤单抗治疗，血脂康组52例给予血脂康胶囊联合依洛尤单抗治疗，两组均连续用药半年。比较两组治疗前、治疗1个月、3个月、6个月后血脂水平，比较两组治疗前、治疗6个月PCSK9、胆固醇酯转移蛋白（cholesterol ester transfer protein，CETP）、载脂蛋白B（apolipoprotein B，ApoB）、心功能、6分钟步行试验、谷丙氨酸转氨酶（glutamic acid aminotransferase，ALT）、谷草转氨酶（glutamine transaminase，AST）、肌酸激酶同工酶（creatine kinase isoenzymes，CK-MB）及中医证候积分水平，统计两组6个月内累计终点事件。
结果 两组治疗前血脂指标水平比较，差异无统计学意义（P>0.05）。两组治疗1个月、3个月、6个月后总胆固醇（total cholesterol，TG）、甘油三酯（triglycerides，TC）、低密度脂蛋白胆固醇（low density lipoprotein cholesterol，LDL-C）水平均较治疗前降低，血脂康组治疗1个月、3个月、6个月后TG、TC、LDL-C水平低于常规组（P<0.05）。两组治疗1个月、3个月、6个月后高密度脂蛋白胆固醇（high density lipoprotein cholesterol，HDL-C）水平比较，差异无统计学意义（P>0.05）。两组治疗前PCSK9、CETP、ApoB、AST、ALT、CK-MB水平比较，差异无统计学意义（P>0.05）。两组治疗6个月后PCSK9、ApoB、AST、ALT、CK-MB水平均较治疗前降低，血脂康组治疗6个月后PCSK9、ApoB水平低于常规组（P<0.05）。两组治疗6个月后CETP、AST、ALT、CK-MB水平比较，差异无统计学意义（P>0.05）。两组治疗前心功能、6分钟步行试验水平比较，差异无统计学意义（P>0.05）。两组治疗6个月后左室射血分数（left ventricular ejection fraction，LVEF）、心排血量（cardiac output，CO）、6分钟步行试验水平较治疗前升高，血脂康组治疗6个月后LVEF、CO、6分钟步行试验水平高于常规组（P<0.05）。两组治疗6个月后左心室舒张末期内径（left ventricular end-diastolic internal diameter，LVESD）水平均较治疗前降低，血脂康组治疗6个月后LVESD水平低于常规组（P<0.05）。两组累计终点事件比较，差异无统计学意义（P>0.05）。治疗前，两组中医证候积分比较，差异无统计学意义（P>0.05）。两组治疗6个月后中医证候积分较治疗前降低，血脂康组治疗低于常规组（P<0.05）。
结论 血脂康联合PCSK9抑制剂治疗他汀不耐受冠心病可调节脂代谢，改善心功能，安全性高。
, authors=吴军
1, 吕晓鹏
2, 张悦清
1, 刘毅
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吴军（1989-），男，山西朔州人，主治医师，硕士，研究方向：冠心病药物治疗。
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吴军（1989-），男，山西朔州人，主治医师，硕士，研究方向：冠心病药物治疗。
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