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Article(id=1240950909354701009, tenantId=1146029695717560320, journalId=1227665162245664772, issueId=1240950898113966774, articleNumber=null, orderNo=null, doi=10.20043/j.cnki.MPM.202309187, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=1694275200000, receivedDateStr=2023-09-10, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1773795352543, onlineDateStr=2026-03-18, pubDate=1712678400000, pubDateStr=2024-04-10, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1773795352543, onlineIssueDateStr=2026-03-18, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1773795352543, creator=13701087609, updateTime=1773795352543, updator=13701087609, issue=Issue{id=1240950898113966774, tenantId=1146029695717560320, journalId=1227665162245664772, year='2024', volume='51', issue='7', pageStart='1153', pageEnd='1344', issueExtLink='null', onlineDate='null', pubDate='null', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=null, createTime=1773795349862, creator=13701087609, updateTime=1773795519367, updator=13701087609, preIssue=null, nextIssue=null, ext={EN=IssueExt(id=1240951609136567133, tenantId=1146029695717560320, journalId=1227665162245664772, issueId=1240950898113966774, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1240951609136567134, tenantId=1146029695717560320, journalId=1227665162245664772, issueId=1240950898113966774, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null}, startPage=1217, endPage=1221, ext={EN=ArticleExt(id=1240950909816074489, articleId=1240950909354701009, tenantId=1146029695717560320, journalId=1227665162245664772, language=EN, title=Research progress on the relationship between dietary inflammation index and sleep quality, columnId=1240950909149171753, journalTitle=Modern Preventive Medicine, columnName=Nutrition Food Hygiene, runingTitle=null, highlight=null, articleAbstract=
Objective

To analyze and summarize the relationship between sleep quality and dietary inflammatory index (DII), so as to provide guidance for improving sleep quality.

Methods

Using dietary inflammatory index, anti-inflammatory diet, anti-inflammatory diet, and sleep quality as keywords, China knowledge net, VIP database, Wan fang database, PubMed and Web of Science database were searched, and the related literatures at home and abroad were reviewed to summarize the relationship between DII and sleep quality and its internal mechanism.

Results

There was a correlation between sleep quality and DII, but the conclusion was not consistent. On one hand, pro-inflammatory or anti-inflammatory diet may affect sleep quality by affecting circadian rhythm system and sleep homeostasis. On the other hand, low-quality sleep may affect dietary selection and intake, thus affecting the level of dietary inflammation.

Conclusion

There is a correlation between sleep quality and DII, but there is no consistent conclusion. Larger samples and high-quality studies are needed in order to provide reference for improving sleep quality.

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目的

分析和总结睡眠质量与膳食炎症指数(dietary inflammatory index,DII)的关系，为改善睡眠质量提供指导。

方法

以膳食炎症指数、抗炎饮食、促炎饮食、睡眠质量等为主题词对中国知网、维普数据库、万方数据库、Pubmed和Web of Science等数据库进行检索，查阅国内外相关文献，总结DII与睡眠质量的关系及其内在机制。

结果

睡眠质量与DII存在关联，但结论尚不统一。一方面促炎或抗炎饮食可能通过影响昼夜节律系统及睡眠稳态对睡眠质量产生影响，另一方面低质量睡眠可能影响膳食的选择与摄入，进而影响膳食炎症水平。

结论

睡眠质量与DII存在关联，但研究尚无统一结论，仍需更多大样本、高质量的研究对其进行进一步探讨，以期为改善睡眠质量提供参考。

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赵梅，E-mail :
, copyrightStatement=本刊刊出的所有文章不代表中华预防医学会和本刊编委会的观点，除非特别声明。, copyrightOwner=中华预防医学会和四川大学华西公共卫生学院, extLink=null, articleAbsUrl=null, sourceXml=EeAA0V8lvMTrZTSjOgC2+w==, magXml=LGBZSzhS8SWtSCzBK54hYA==, pdfUrl=null, pdf=wD0ZENf6qBswxWBq4UPBHA==, pdfFileSize=696133, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=+ll2zS6aT/AbizHQHjAU3g==, mapNumber=null, authorCompany=null, fund=null, authors=

张锦茹（1999—），女，硕士在读，研究方向：营养与健康

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张锦茹（1999—），女，硕士在读，研究方向：营养与健康

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Definition and standards of sleep quality related indicators

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睡眠质量指标定义及标准
睡眠潜伏期睡眠潜伏期指的是从躺下就寝到开始睡眠的时间，主要反映入睡状况，正常应在20～30 min，睡眠潜伏期越长说明入睡难度越大
唤醒时间唤醒时间(wake-after-sleep-onset ,WASO)指的是睡眠开始后直到睡眠结束或被唤醒后的醒着分钟数的总和，为了避免睡眠期间肢体的周期性运动，每个醒着的时间必须至少2 min才能计入WASO
睡眠时长睡眠时长是指从入睡期开始到快速眼动期结束时的时间。美国睡眠基金会建议18～64岁人群最佳夜间睡眠时长为7～9 h，65岁及以上人群最佳夜间睡眠时间为7～8 h，当个体睡眠时长小于7h/晚时被认为存在睡眠不足
睡眠效率睡眠效率的计算方法为躺在床上的总时间除以睡眠潜伏期、睡眠持续时间和WASO的总和，通常大于0.8表明睡眠质量佳
睡眠障碍睡眠障碍被定义为睡眠量或质的异常，或睡眠时发生睡眠时间缩短、深睡眠持续时间减少、夜间觉醒次数增多、醒后难以入睡及早醒等临床症状
), ArticleFig(id=1240972171439305509, tenantId=1146029695717560320, journalId=1227665162245664772, articleId=1240950909354701009, language=CN, label=表1, caption=

睡眠质量相关指标定义及标准

, figureFileSmall=null, figureFileBig=null, tableContent=
睡眠质量指标定义及标准
睡眠潜伏期睡眠潜伏期指的是从躺下就寝到开始睡眠的时间，主要反映入睡状况，正常应在20～30 min，睡眠潜伏期越长说明入睡难度越大
唤醒时间唤醒时间(wake-after-sleep-onset ,WASO)指的是睡眠开始后直到睡眠结束或被唤醒后的醒着分钟数的总和，为了避免睡眠期间肢体的周期性运动，每个醒着的时间必须至少2 min才能计入WASO
睡眠时长睡眠时长是指从入睡期开始到快速眼动期结束时的时间。美国睡眠基金会建议18～64岁人群最佳夜间睡眠时长为7～9 h，65岁及以上人群最佳夜间睡眠时间为7～8 h，当个体睡眠时长小于7h/晚时被认为存在睡眠不足
睡眠效率睡眠效率的计算方法为躺在床上的总时间除以睡眠潜伏期、睡眠持续时间和WASO的总和，通常大于0.8表明睡眠质量佳
睡眠障碍睡眠障碍被定义为睡眠量或质的异常，或睡眠时发生睡眠时间缩短、深睡眠持续时间减少、夜间觉醒次数增多、醒后难以入睡及早醒等临床症状
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膳食炎症指数与睡眠质量关系的研究进展
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张锦茹 , 洪丽 , 赵梅
现代预防医学 | 营养与食品卫生 2024,51(7): 1217-1221
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现代预防医学 | 营养与食品卫生 2024, 51(7): 1217-1221
膳食炎症指数与睡眠质量关系的研究进展
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张锦茹, 洪丽, 赵梅
作者信息
  • 安徽医科大学护理学院，安徽 合肥 230032

    • 张锦茹（1999—），女，硕士在读，研究方向：营养与健康

通讯作者:

赵梅，E-mail :
Research progress on the relationship between dietary inflammation index and sleep quality
Jin-ru ZHANG, Li HONG, Mei ZHAO
Affiliations
  • College of Nursing, Anhui Medical University, Hefei, Anhui 230032, China
出版时间: 2024-04-10 doi: 10.20043/j.cnki.MPM.202309187
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摘要
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目的

分析和总结睡眠质量与膳食炎症指数(dietary inflammatory index,DII)的关系，为改善睡眠质量提供指导。

方法

以膳食炎症指数、抗炎饮食、促炎饮食、睡眠质量等为主题词对中国知网、维普数据库、万方数据库、Pubmed和Web of Science等数据库进行检索，查阅国内外相关文献，总结DII与睡眠质量的关系及其内在机制。

结果

睡眠质量与DII存在关联，但结论尚不统一。一方面促炎或抗炎饮食可能通过影响昼夜节律系统及睡眠稳态对睡眠质量产生影响，另一方面低质量睡眠可能影响膳食的选择与摄入，进而影响膳食炎症水平。

结论

睡眠质量与DII存在关联，但研究尚无统一结论，仍需更多大样本、高质量的研究对其进行进一步探讨，以期为改善睡眠质量提供参考。

膳食炎症指数  /  睡眠质量  /  作用机制
Objective

To analyze and summarize the relationship between sleep quality and dietary inflammatory index (DII), so as to provide guidance for improving sleep quality.

Methods

Using dietary inflammatory index, anti-inflammatory diet, anti-inflammatory diet, and sleep quality as keywords, China knowledge net, VIP database, Wan fang database, PubMed and Web of Science database were searched, and the related literatures at home and abroad were reviewed to summarize the relationship between DII and sleep quality and its internal mechanism.

Results

There was a correlation between sleep quality and DII, but the conclusion was not consistent. On one hand, pro-inflammatory or anti-inflammatory diet may affect sleep quality by affecting circadian rhythm system and sleep homeostasis. On the other hand, low-quality sleep may affect dietary selection and intake, thus affecting the level of dietary inflammation.

Conclusion

There is a correlation between sleep quality and DII, but there is no consistent conclusion. Larger samples and high-quality studies are needed in order to provide reference for improving sleep quality.

Dietary inflammation index  /  Sleep quality  /  Mechanism
张锦茹, 洪丽, 赵梅. 膳食炎症指数与睡眠质量关系的研究进展. 现代预防医学, 2024 , 51 (7) : 1217 -1221 . DOI: 10.20043/j.cnki.MPM.202309187
Jin-ru ZHANG, Li HONG, Mei ZHAO. Research progress on the relationship between dietary inflammation index and sleep quality[J]. Modern Preventive Medicine, 2024 , 51 (7) : 1217 -1221 . DOI: 10.20043/j.cnki.MPM.202309187
正文
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睡眠是保障机体身心健康的重要方式，高质量的睡眠对于正常脑功能、系统生理、免疫调剂等方面都具有至关重要的作用[1]。然而当前我国有近30%的成年人存在睡眠问题，睡眠质量低已逐渐成为一个公共卫生问题[2]。睡眠质量(sleep quality)是个体主观与客观睡眠指标的综合反映，代表个体睡眠的好坏程度，睡眠质量既包括睡眠的定量方面(如睡眠潜伏期或觉醒次数、睡眠时长等)，也包括睡眠的定性方面(如睡眠的感受、深度等)[3]。现有研究表明，睡眠质量可能与机体炎症水平有关，一方面，白细胞介素-6(interleukin-6, IL-6)、C反应蛋白(C-reactive protein ,CRP)、肿瘤坏死因子-α(tumor necrosis factor-alpha,TNF-α)等炎症标记物水平的升高会引起睡眠质量的下降[4]；另一方面，睡眠质量低也可能引起机体炎症水平改变[5-6]。作为促炎或抗炎化合物的重要潜在来源，饮食是炎症的重要调节工[7]。膳食炎症指数(dietary inflammatory index,DII)是文献衍生的膳食工具，可以用来评估个体饮食的总体炎症潜能[8]。当前越来越多的研究针对DII与睡眠质量之间的关系进行探索，基于此，本文就DII与睡眠质量之间的关系及内在机制进行综述，旨在为缓解睡眠问题、提高睡眠质量提供科学依据及参考。
1　膳食炎症指数概述
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1.1　DII的提出与优化
哥伦比亚南卡罗莱纳大学学者通过检索、筛选1950—2007年间有关炎症与食物成分关系的文章来评估食物成分对特定炎症标记物的影响(如CRP、IL-1β、IL-6和TNF-α等)，结合细胞培养、动物实验和流行病学研究，于2009年开发并验证了初版DII[8]。2013年van Woudenbergh等[9]学者对能量、脂肪摄入、膳食成分消耗的变化及乙醇含量进行调整，提出适应性膳食炎症指数(adapted dietary inflammatory index,ADII)。2014年Shivappa N等[10]学者更新同行评审文章，将饮食参数列表扩展至45个项目，并汇总全球11个食品消费数据库完善了DII的评分算法。2016年，在护士健康研究数据以及食物和炎症相关知识的基础上，Tabung等[11]学者开发了基于不同类别食物组计算膳食炎症潜能的经验性膳食炎症指数(empirical dietary inflammatory index, EDII)，较DII而言EDII对于指导膳食更具实际应用价值且直观。
鉴于饮食习惯和饮食文化的差别，我国居民膳食模式与西方国家截然不同。为评估中国人群膳食炎症潜力，中国学者构建了一套适合中国人群的膳食炎症评估工具。2021年，为评估中国孕妇的膳食炎症潜力，张珍[12]开发并验证了中国孕妇膳食炎症指数(Chinese pregnant women’s dietary inflammation index, CPW-DII)。2022年，基于北京昌平农村社区2型糖尿病管理项目，吕璐[13]构建了中国经验性炎症膳食指数(Chinese empirical dietary inflammatory index,CEDII)，这也是第一个基于我国居民饮食数据开发的膳食炎症潜力评估工具。
作为膳食炎症潜力的评估工具，DII可帮助了解个体的饮食质量。当前，DII逐渐被应用在慢性病、癌症等疾病的研究与防治中[14-16]。
1.2　DII与炎症的关系
饮食是机体炎症水平的重要影响因素，DII的开发为膳食营养的炎症风险提供了定量评价的标准[8]。DII可以同时捕捉个体摄入的多种膳食成分的综合炎症效应，克服了单个膳食成分或营养素难以总结的缺陷，DII评分越高表明膳食对机体的促炎作用越强[10]。
DII评分较高(即饮食较促炎)的个体CRP、IL-6、白细胞、中性粒细胞/淋巴细胞比率(NLR)水平均较高[8]。一方面，促炎饮食可以诱发机体炎症细胞因子大量激活，还可导致细胞增殖、炎症和氧化应激增加。精制碳水化合物、饱和脂肪、高糖饮料、油炸食品、白酒和ω-6脂肪酸等食物成分被证实具有促炎性，可显著增加全身炎症生物标志物，如CRP、IL-6和白细胞介素10(IL-10)[17]。另一方面，促炎饮食会改变肠道微生物生态系统，引起肠道屏障功能障碍、肠道通透性增加以及有毒代谢物向循环系统泄露，促进慢性炎症的发生和发展[18]。例如高饱和脂肪可以诱发肠道菌群失调，激活肠道炎症信号通路，还易引起肠道屏障完整性损伤，导致细菌或脂多糖等菌体成分移位，诱发系统性炎症[19]。作为一种促炎饮食，西方饮食与较高的炎症标记物水平有关。西方饮食的特点是摄入大量加工、精制、油炸类食物如精制糖(包括糖果、甜食和软饮料等)、动物脂肪(摄入大量饱和脂肪酸和ω-6脂肪酸，减少ω-3脂肪酸摄入)、加工肉类、精制谷物、高脂乳制品等，而水果、蔬菜、全谷物、鱼、坚果等摄入较少。研究发现，西方饮食会改变胆固醇代谢并导致低度炎症的发生和发展，表现为循环促炎因子的升高[20]。
与促炎饮食相比，抗炎饮食可以通过降低机体炎症生物标记物水平发挥抑制机体炎症发生和发展的作用[21]。鱼类、酸奶、大米、水果、绿叶蔬菜，坚果已被证明可以降低炎症生物标志物水平[22]。膳食纤维是一种典型的抗炎营养素，其可以促进肠道有益共生菌的生长，增加菌群多样性，帮助机体免疫稳态的维持[23]。地中海饮食是一种抗炎饮食，对肠道菌群有积极影响，其特点是大量摄入蔬菜、水果、谷物(主要是全谷物)、坚果和豆类，较少摄入甜食、肉类和不饱和脂肪酸。坚持地中海饮食与较低的CRP、白细胞水平有关，此外坚持地中海饮食还可使多种疾病的患病率降低并减少炎症的发和与发展[21]。
2　睡眠质量与DII
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Nelson等[3]学者于2022年对睡眠质量进行概念分析，提出睡眠质量是个体对睡眠体验的自我满意度，可以通过睡眠潜伏期、唤醒时间、睡眠时长、睡眠效率及睡眠障碍来衡量，见表1。睡眠质量的评估方式包括客观测量和使用主观评估工具两种方法，当前有关DII和睡眠质量的研究中主要采用匹兹堡睡眠质量指数对睡眠质量进行评估，PSQI评分超过5分被认为是睡眠质量差[3]。
当前针对DII与睡眠质量的关系存在一定争议。部分研究发现DII得分高与睡眠质量低有关。Justyna Godos等[24]学者对意大利成年人的DII与睡眠质量的关系进行调查，结果显示，DII得分较高的个体睡眠质量可能较低。在一项针对伊朗女学生的研究中也发现了同样的结论，Hadi Bazyar等[25]学者对249名女大学生进行调查，在排除混杂因素影响后，DII得分最高的四分位数的参与者（即饮食最促炎）PSQI得分较高（即睡眠质量低）。此外，还有研究发现抗炎饮食可能会改善成年人的睡眠质量，个体饮食质量提高，睡眠质量也会提高[26-27]。然而Masaad等[28]学者对379名大学生进行调查，未在DII与睡眠质量之间发现显著关联，但在较高DII四分位数组中，睡眠质量低的人比例较高。在一项针对警察的研究中，随着DII分数的增加(即促炎性更强)，PSQI整体睡眠评分会下降(即睡眠改善)，且从类别角度而言，促炎类别个体PSQI均数低于抗炎类别个体，这表明，DII得分高可能与睡眠质量改善有关[29]。
在DII与睡眠质量相关指标方面，现有研究发现，当DII增加时(即饮食变得更促炎时)，个体睡眠潜伏期与唤醒时间增加、睡眠效率降低[29]。此外与抗炎组相比，促炎组调查对象更有可能报告存在睡眠障碍[29-30]。然而有关DII和睡眠时长的关系目前尚未统一。一项对211名医科大学不同部门的员工饮食质量与睡眠关系的调查发现，DII与睡眠时长呈显著负相关[29]。Michael D. Wirth等[29]学者发现与最低五分之一组(最具抗炎性的DII)相比，最高五分之一组(最具促炎性的DII)参与者睡眠不足几率增加，此外饮食越促炎，睡眠时间过长的几率也会增加。舒艳铃等[30]学者利用美国健康与营养调查数据库分析美国老年人DII与睡眠时长的关系，结果显示，DII与睡眠时长之间存在非线性关系，且DII每增加1个单位,睡眠不足发生可能性提高6.3%。还有研究发现DII和睡眠时长之间无明显关联[31]。
这些矛盾的研究结果可能与多种因素有关。一方面，不同人群睡眠模式存在差异。通过全面的睡眠质量评估（包括采用多导睡眠图、活动描述图、相关评估问卷进行睡眠质量评估）发现，不同种族的人群睡眠质量存在差异，其中睡眠时长差异最为显著[32]。另一方面，DII多采用食物频率问卷、24小时膳食回顾或访谈等评估个人膳食摄入量，不同评估方式以及研究者是否接受过专业培训对调查资料的准确性、全面性可能产生影响[33]，这会进一步影响研究的结果。因此，睡眠质量与DII的相关性还需更大规模，涵盖不同种族、不同饮食习惯人群的高质量研究加以验证。
3　睡眠质量与DII的可能机制
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为进一步分析睡眠质量与DII的关系，需要对其内在机制进行探讨。现有研究表明，睡眠和饮食存在双向关系[34]。由于目前针对睡眠质量与DII的研究多为横断面调查，无法展现两者之间的因果关系，因此本文拟从两方面分析睡眠质量与DII之间可能存在的内在机制。
3.1　DII影响睡眠质量的机制
睡眠是一个动态的过程，昼夜节律系统和睡眠稳态两个因素通过相互作用整合调节睡眠需求[35]。昼夜节律对睡眠觉醒行为的调节反映了外界因素对觉醒的需求，睡眠稳态调节反映机体内部因素对睡眠的需求[36]。
现有研究表明饮食会影响昼夜节律功能，促炎潜力高的饮食会影响昼夜节律基因，并编码重要的基因代谢环境[37-38]。目前尚不清楚其内在机制，但一些动物实验结果表明，表观遗传学可能是一种可能的机制。研究发现，高脂肪和高碳水化合物饮食可以降低烟酰胺腺嘌呤二核苷酸水平，并降低乙酰辅酶A含量，同时提高长链游离脂肪酸，增加SIRT6脱乙酰酶活性的激活，从而影响昼夜节律代谢过程[38-39]。
此外，饮食还可对睡眠稳态产生影响。Zuraikat等[40]发现，水果蔬菜摄入越多，睡眠质量越高。蔬菜、水果、谷物等抗炎饮食含有丰富的氨基丁酸，而氨基丁酸可以抑制中枢神经系统活动，帮助个体缩短入睡潜伏期，延长睡眠。此外，作为一种公认的睡眠诱导剂，褪黑素往往也存在于水果、蔬菜、全谷物等食物中，其对睡眠的启动、维持和睡眠期间的稳态恢复同样具有重要作用[41]。褪黑素通过激活褪黑素1型(melatonin type 1 receptor receptor, MT1R)和2型受体(MT2R)发挥调控睡眠的作用[42]。还有一些维生素，如B12，可以促进褪黑激素的生成，从而提高睡眠质量[43]。一些促进睡眠的细胞因子，如IL-6、IL-1β和TNF-α，这几种细胞因子都具有促炎性，往往和一些促炎饮食相关。IL-1β可以刺激生长激素释放激素，从而增强非快速动眼睡眠[1]。IL-6与睡眠开始有关，当睡眠开始时其达到峰值[44]。长期摄入促炎饮食会增加这些细胞因子的水平，影响其分泌节律，破坏睡眠结构[44-45]。
饮食还可通过影响机体炎症水平从而影响睡眠质量。极端的睡眠时长(睡眠不足或睡眠过长)与促炎因子浓度增加有关[46]。摄入大量肉制品、精制碳水化合物或超加工食品与促炎反应增加有关，这可能降低睡眠质量[47]。低炎症潜能的饮食则与更高的睡眠质量有关[46]。ω-3和单不饱和脂肪酸都具有抗炎特性，研究发现十二碳六烯酸或二十碳六烯酸与花生四烯酸之间的比例均会影响炎症水平，从而影响睡眠质量[25,48]。
3.2　睡眠质量影响DII的机制
多项研究表明，睡眠对饮食的选择及摄入具有重要作用，极端睡眠时长(睡眠时间过长或过短)和睡眠质量差和不良饮食行为有关[49]。实验表明，睡眠不足会导致摄入食物种类发生变化，睡眠不足的个体会增加脂肪与碳水化合物的摄入，并促进零食的消费，减少蔬菜、水果和全谷物的摄入，降低整体饮食质量[50]。Yasmin等[51]学者采用2010年健康饮食指数(HEI-2010)对西班牙裔/拉丁裔成年人进行饮食质量调查，结果表明，睡眠时间过长与低质量饮食相关。一方面，昼夜节律失调的睡眠限制会增加胃饥饿素、瘦素、胰高血糖素养肽1(GLP-1)和食欲素等食欲调节激素的分泌，诱发个体对不健康饮食的摄入，导致膳食炎症水平提高[52]。另一方面，睡眠质量低可能增加个体对食物的享乐驱动力。低质量的睡眠可能引起食物奖励及杏仁核和下丘脑活动的主观评价选择性增加[53]。研究表明，睡眠不足会侵害个体的执行能力，减少控制和抑制相关的电活动，致使饮食方面的自制力减弱，进而诱发暴饮暴食，降低饮食质量[54]。
4　讨论
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近年来，睡眠质量下降逐渐成为一个公共卫生问题，饮食可能是调节睡眠质量的重要因素。作为评判个体膳食炎症水平的工具，DII被越来越多的应用在饮食与睡眠关系的研究中。但当前，受研究设计、研究人群选择等因素影响，目前相关结论并不一致，且当前睡眠质量与DII的内在机制也仍不明晰。结合现有研究，本文拟提出以下展望：（1）当前有关DII与睡眠质量的文章多为横断面研究，尚不能得出两者的因果关系，仍需更多大样本、高质量的研究探究其内在关联；（2）目前探讨DII与睡眠质量的内在机制的研究多从单一营养素或常见饮食模式(如地中海饮食、西方饮食等)出发，未来可从抗炎类饮食和促炎类饮食角度出发，进一步探讨DII与睡眠质量的内在机制。
基金
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  • 安徽省高校人文社会科学研究项目重大项目(SK2021ZD0030)
  • 2023安徽医科大学护理学院研究生青苗培育项目(hlqml12023062)
文献
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doi: 10.20043/j.cnki.MPM.202309187
  • 接收时间：2023-09-10
  • 首发时间：2026-03-18
  • 出版时间：2024-04-10
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  • 收稿日期：2023-09-10
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安徽省高校人文社会科学研究项目重大项目(SK2021ZD0030)
2023安徽医科大学护理学院研究生青苗培育项目(hlqml12023062)
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    安徽医科大学护理学院，安徽 合肥 230032

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赵梅，E-mail :
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2种不同金属材料的力学参数
科
Family		 属数
Number of
genus		 种数
Number of
species		 占总种数比例
Percentage of
total species (%)		 属
Genus		 种数
Number of
species		 占总种数比例
Percentage of total
species (%)
鹅膏菌科Amanitaceae 2 11 5.26 鹅膏菌属 Amanita 10 4.78
小菇科 Mycenaceae 2 12 5.74 丝盖伞属 Inocybe 5 2.39
多孔菌科 Polyporaceae 8 14 6.70 蜡蘑属 Laccaria 5 2.39
红菇科 Russulaceae 3 23 11.00 小皮伞属 Marasmius 6 2.87
小菇属 Mycena 11 5.26
光柄菇属 Pluteus 5 2.39
红菇属 Russula 17 8.13
栓菌属 Trametes 5 2.39
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