Article(id=1315688510493938286, tenantId=1146029695717560320, journalId=1315121837170065538, issueId=1315688435411694395, articleNumber=null, orderNo=null, doi=10.16368/j.issn.1674-8999.2025.12.405, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=1750089600000, receivedDateStr=2025-06-17, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1791614185211, onlineDateStr=2026-10-10, pubDate=1765296000000, pubDateStr=2025-12-10, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1791614185211, onlineIssueDateStr=2026-10-10, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1791614185211, creator=13701087609, updateTime=1791614185211, updator=13701087609, issue=Issue{id=1315688435411694395, tenantId=1146029695717560320, journalId=1315121837170065538, year='2025', volume='40', issue='12', pageStart='2487', pageEnd='2712', issueExtLink='null', onlineDate='null', pubDate='1765296000000', pubDateStr='2025-12-10', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=null, createTime=1791614167309, creator='13701087609', updateTime=1791616084449, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1315696476567744680, tenantId=1146029695717560320, journalId=1315121837170065538, issueId=1315688435411694395, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1315696476571938985, tenantId=1146029695717560320, journalId=1315121837170065538, issueId=1315688435411694395, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=2588, endPage=2594, ext={EN=ArticleExt(id=1315688510733013615, articleId=1315688510493938286, tenantId=1146029695717560320, journalId=1315121837170065538, language=EN, title=Research Progress of Traditional Chinese Medicine in Treatment of Atherosclerosis, columnId=1315688445339612089, journalTitle=Acta Chinese Medicine, columnName=Commentary, runingTitle=null, highlight=null, articleAbstract=

Atherosclerosis（AS）is fundamentally rooted in Qi deficiency，with phlegm and blood stasis as its key pathological products. The mutual entanglement of phlegm and blood stasis leading to vascular obstruction is its main pathological manifestation.Effective monomers，herbal pairs，and compound prescriptions of traditional Chinese medicine that tonify Qi，resolve phlegm，activate blood，and unblock collaterals can intervene in the progression of AS through mechanisms such as anti-inflammation，antioxidation，regulation of lipid metabolism，inhibition of cell apoptosis，and improvement of vascular endothelial function，demonstrating the significant advantages of traditional Chinese medicine in the prevention and treatment of AS.The method of benefiting Qi，resolving phlegm，and removing stasis serves as a potential strategy for early intervention in AS and plays an important role in preventing complications such as cardiovascular and cerebrovascular diseases.In the future，the methodology and evaluation system for integrating traditional Chinese medicine（TCM）syndromes with animal models should be further improved. By incorporating findings from modern medical research，the mechanisms of″Qi deficiency，Phlegm and blood stasis″should be explored in depth.At the same time，multicenter，large -sample clinical trials should be conducted to uncover the correlation rules between TCMsyndromes and modern medicine，aiming to provide objective evidence and new insights for the early prevention of atherosclerosis，drug development，optimization of TCM diagnostic and treatment standards，and individualized therapy.

, authors=Xiaoxiao FU, Shan CAO, Xiang QI, authorsList=Xiaoxiao FU, Shan CAO, Xiang QI, authorCompany=null, correspAuthors=Shan CAO, authorNote=null, correspAuthorsNote=null, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1315688511131472496, articleId=1315688510493938286, tenantId=1146029695717560320, journalId=1315121837170065538, language=CN, title=中医药治疗动脉粥样硬化研究进展, columnId=1315688445511578555, journalTitle=中医学报, columnName=述评, runingTitle=null, highlight=null, articleAbstract=

动脉粥样硬化（atherosclerosis，AS）以气虚为本，痰、瘀为其关键病理产物，痰瘀互结致血脉瘀阻为其主要病理表现。补气、化痰、活血、通络类中药有效单体、药对配伍及复方均可通过抗炎、抗氧化、调节脂代谢、抑制细胞凋亡及改善血管内皮功能等机制干预AS进展，显示出中医药在防治AS方面的显著优势。益气化痰祛瘀法作为早期干预AS的潜在策略，在预防心脑血管疾病等并发症方面具有重要作用。未来应进一步完善中医病证结合动物模型的构建方法与评价体系，结合现代医学研究成果，深入阐释“气虚痰瘀”的机制；同时开展多中心、大样本临床试验，发掘中医证候与现代医学之间的关联规律，以期为AS的早期预防、药物研发、中医诊疗标准体系优化及个体化治疗提供客观依据与新思路。

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付晓霄（1999 -），女，河南郑州人，硕士研究生，研究方向：方剂配伍规律与作用机制研究。

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付晓霄（1999 -），女，河南郑州人，硕士研究生，研究方向：方剂配伍规律与作用机制研究。

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