Article(id=1276975973090324673, tenantId=1146029695717560320, journalId=1276577828834349082, issueId=1276975955985961244, articleNumber=null, orderNo=null, doi=10.19540/j.cnki.cjcmm.20250929.801, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=1756656000000, receivedDateStr=2025-09-01, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1782384397050, onlineDateStr=2026-06-25, pubDate=1767196800000, pubDateStr=2026-01-01, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1782384397050, onlineIssueDateStr=2026-06-25, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1782384397050, creator=13701087609, updateTime=1782384397050, updator=13701087609, issue=Issue{id=1276975955985961244, tenantId=1146029695717560320, journalId=1276577828834349082, year='2026', volume='51', issue='1', pageStart='1', pageEnd='300', issueExtLink='null', onlineDate='null', pubDate='1767196800000', pubDateStr='2026-01-01', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=null, createTime=1782384392973, creator='13701087609', updateTime=1782384484405, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1276976339546673871, tenantId=1146029695717560320, journalId=1276577828834349082, issueId=1276975955985961244, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1276976339546673872, tenantId=1146029695717560320, journalId=1276577828834349082, issueId=1276975955985961244, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=50, endPage=55, ext={EN=ArticleExt(id=1276975973530726595, articleId=1276975973090324673, tenantId=1146029695717560320, journalId=1276577828834349082, language=EN, title=TCM regulates endothelial-to-mesenchymal transition in treatment of atherosclerosis: a review, columnId=null, journalTitle=Chinese Journal of Traditional Chinese Medicine, columnName=null, runingTitle=null, highlight=null, articleAbstract=

Endothelial-to-mesenchymal transition (EndMT) is a process in which endothelial cells lose specific markers and acquire a mesenchymal phenotype under pathological stimulation, contributing to the occurrence and progression of atherosclerosis (AS). Recent studies have shown that TCM, with its unique advantages of holism and treatment based on syndrome differentiation, can inhibit AS by regulating EndMT-related signaling pathways at multiple levels, through multiple pathways and targets. This article systematically reviews the mechanisms of EndMT in AS, as well as the research progress of TCM monomers and compounds in counteracting AS via EndMT intervention, providing new ideas and a scientific basis for the prevention and treatment of AS with TCM.

, authors=null, authorsList=Xue-yan ZHANG, Ying-zhu DUAN, You YU, Huan ZHANG, Jiao-dan WANG, Xin-ran GENG, Rui YU, authorCompany=null, correspAuthors=Rui YU, authorNote=null, correspAuthorsNote=null, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1276975974071791813, articleId=1276975973090324673, tenantId=1146029695717560320, journalId=1276577828834349082, language=CN, title=中医药调节血管内皮细胞间质转化抗动脉粥样硬化研究进展, columnId=1276975957151977760, journalTitle=中国中药杂志, columnName=综述, runingTitle=null, highlight=null, articleAbstract=

内皮间质转化（endothelial-to-mesenchymal transition, EndMT）是内皮细胞在病理刺激下失去特异性标志物、获得间质细胞表型的转化过程，参与动脉粥样硬化（atherosclerosis, AS）的发生与发展。近年研究发现，中医药凭借其整体观、辨证论治等独特优势，可通过多层次、多途径、多靶点调控EndMT相关信号通路抑制AS进程。该文系统综述EndMT在AS中的作用机制，以及中药单体和复方通过干预EndMT抗AS的研究进展，为中医药防治AS提供新的思路和研究依据。

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张雪妍，博士研究生，主要从事中西医结合防治心血管疾病的研究，E-mail: m13523602561@163.com

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张雪妍，博士研究生，主要从事中西医结合防治心血管疾病的研究，E-mail: m13523602561@163.com

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