Article(id=1292135509623591218, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292135444850955150, articleNumber=null, orderNo=null, doi=10.13699/j.cnki.1001-6821.2026.09.019, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=1769961600000, receivedDateStr=2026-02-02, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1785998712210, onlineDateStr=2026-08-06, pubDate=1778947200000, pubDateStr=2026-05-17, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1785998712210, onlineIssueDateStr=2026-08-06, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1785998712210, creator=13701087609, updateTime=1785998712210, updator=13701087609, issue=Issue{id=1292135444850955150, tenantId=1146029695717560320, journalId=1246415772164075586, year='2026', volume='42', issue='9', pageStart='1201', pageEnd='1350', issueExtLink='null', onlineDate='null', pubDate='1778947200000', pubDateStr='2026-05-17', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=null, createTime=1785998696767, creator='13701087609', updateTime=1786014392260, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1292201276780081469, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292135444850955150, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1292201276784275774, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292135444850955150, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=1323, endPage=1329, ext={EN=ArticleExt(id=1292135511217426739, articleId=1292135509623591218, tenantId=1146029695717560320, journalId=1246415772164075586, language=EN, title=Research status on the mechanism of microRNA in regulating heart failure and the intervention of traditional Chinese medicine, columnId=1246531408630534361, journalTitle=Chinese Journal of Clinical Pharmacology, columnName=Review, runingTitle=null, highlight=null, articleAbstract=

Heart failure (HF) refers to a clinical syndrome caused by structural or functional cardiac disorders that impair ventricular filling and/or ejection function, resulting in insufficient cardiac output to meet the metabolic demands of bodily tissues. Its clinical manifestations include congestion in the pulmonary and/or systemic circulation, as well as inadequate blood perfusion to organs and tissues. As the terminal stage of various cardiovascular diseases, HF ranks among the leading causes of death for urban and rural residents in China. microRNA (miRNA) is a class of endogenous non-coding single-stranded ribonucleic acids (RNAs) that regulate gene expression at the post-transcriptional level. They are involved in multiple pathological processes of HF, such as myocardial remodeling, cell death, energy metabolism and inflammatory immunity, thus exhibit potential as therapeutic targets for the disease. Traditional Chinese medicine (TCM), characterized by multi-targeted and multi-pathway actions, has shown unique advantages in the prevention and treatment of HF by modulating miRNAs. This article reviews the recent research status on miRNA-mediated regulation of HF and the mechanisms underlying TCM interventions, with the aim of providing insights for subsequent HF-related studies.

, authors=Bao-mei XU¹, Zhen-yu YANG¹, Li-ping YANG¹, Xiao-peng ZHAO², Li ZHANG¹, authorsList=Bao-mei XU, Zhen-yu YANG, Li-ping YANG, Xiao-peng ZHAO, Li ZHANG, authorCompany=null, correspAuthors=Xiao-peng ZHAO, authorNote=null, correspAuthorsNote=null, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1292135511578136884, articleId=1292135509623591218, tenantId=1146029695717560320, journalId=1246415772164075586, language=CN, title=microRNAS调控心衰的机制及中医药干预研究现状, columnId=1246531408819278046, journalTitle=中国临床药理学杂志, columnName=综述, runingTitle=null, highlight=null, articleAbstract=

心力衰竭（HF）是指心脏结构或功能性疾病导致心室充盈和或射血功能受损，心排出量不能满足机体组织代谢需要，以肺循环和或体循环淤血，器官、组织血液灌注不足为临床表现的一组综合征，是多种心血管疾病发展的终末阶段，是我国城乡居民主要的死亡原因之一。微小核糖核酸（microRNA）是一类内源性非编码单链脱氧核糖核酸（RNA），在转录后水平调控基因的表达，参与心肌重构、细胞死亡、能量代谢、炎症免疫等心衰的多种病理进程，展现出作为疾病治疗靶点的潜力。中药（TCM）具有多靶点、多途径的作用特点，microRNA调控在HF防治中展现出独特优势，本文就近年来microRNA调控HF的研究现状及中药干预机制作出综述，希望为HF相关后续研究有所帮助。

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许宝妹（2002-），女，硕士研究生，主要从事中西医结合防治心血管疾病方面的研究

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许宝妹（2002-），女，硕士研究生，主要从事中西医结合防治心血管疾病方面的研究

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