Article(id=1292130379796861541, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292130366320562708, articleNumber=null, orderNo=null, doi=10.13699/j.cnki.1001-6821.2026.07.018, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=1773158400000, receivedDateStr=2026-03-11, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1785997489164, onlineDateStr=2026-08-06, pubDate=1776355200000, pubDateStr=2026-04-17, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1785997489164, onlineIssueDateStr=2026-08-06, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1785997489164, creator=13701087609, updateTime=1785997489164, updator=13701087609, issue=Issue{id=1292130366320562708, tenantId=1146029695717560320, journalId=1246415772164075586, year='2026', volume='42', issue='7', pageStart='901', pageEnd='1050', issueExtLink='null', onlineDate='null', pubDate='1776355200000', pubDateStr='2026-04-17', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=null, createTime=1785997485952, creator='13701087609', updateTime=1786014433453, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1292201449350516861, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292130366320562708, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1292201449350516862, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292130366320562708, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=1011, endPage=1018, ext={EN=ArticleExt(id=1292130380195320422, articleId=1292130379796861541, tenantId=1146029695717560320, journalId=1246415772164075586, language=EN, title=Semaglutide combined with metformin and Traditional Chinese Medicine in the treatment of obese polycystic ovary syndrome: An integrated perspective of Chinese and Western Medicine, columnId=1246531408630534361, journalTitle=Chinese Journal of Clinical Pharmacology, columnName=Review, runingTitle=null, highlight=null, articleAbstract=
Polycystic ovary syndrome (PCOS) is a highly prevalent endocrine and metabolic disorder among women of childbearing age, with insulin resistance (IR) and abnormal glucose and lipid metabolism acting as the core driving factors in obese PCOS. Currently, Western medical therapies represented by metformin combined with glucagon-like peptide-1 receptor agonists (GLP-1RA) (such as semaglutide) have demonstrated definite efficacy in weight loss and IR improvement. However, they are limited by adverse gastrointestinal reactions and a high risk of weight rebound after drug withdrawal. Traditional Chinese Medicine (TCM) posits that the pathogenesis of this disease is characterized by "kidney deficiency as the root, spleen deficiency as the pivot, and phlegm-dampness and blood stasis as the branch." TCM possesses multi-target advantages in regulating body metabolism and reproductive endocrinology. Studies have shown that the synergistic application of TCM formulas combined with appropriate techniques, such as acupuncture and acupoint catgut embedding, alongside the aforementioned Western medicines, can not only further enhance weight loss and metabolic improvement by synergistically regulating key signaling pathways like adenosine 5′-monophosphate-activated protein kinase (AMPK) and phosphatidylinositol 3-kinase/protein kinase B (PI3K/Akt), but also effectively optimize the ovarian microenvironment, improve endometrial receptivity, and significantly antagonize the gastrointestinal adverse reactions induced by Western medicines. This article systematically reviews the core mechanisms, evidence-based data, and safety profile of the combined treatment of metformin, semaglutide, and TCM for obese PCOS. It aims to provide a scientific theoretical basis for formulating standardized integrated Traditional Chinese and Western Medicine treatment protocols and offer new insights to break through the bottlenecks in the long-term clinical management of this disease.
, authors=Chun-you HUANG
1, Yao-qi WEN
2, Hai-yan MENG
1, Bi-xia WANG
1, Dan-hua QIU
1, Hua-mei LI
1, Xiao-li CHEN
1, Ming-mei WANG
1, Shao-yun LI
1, Yu-guan WEN
2, authorsList=Chun-you HUANG, Yao-qi WEN, Hai-yan MENG, Bi-xia WANG, Dan-hua QIU, Hua-mei LI, Xiao-li CHEN, Ming-mei WANG, Shao-yun LI, Yu-guan WEN, authorCompany=null, correspAuthors=Yu-guan WEN, authorNote=null, correspAuthorsNote=null, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1292130381801738857, articleId=1292130379796861541, tenantId=1146029695717560320, journalId=1246415772164075586, language=CN, title=司美格鲁肽联合二甲双胍及中医药治疗肥胖型多囊卵巢综合征:中西医整合视角, columnId=1246531408819278046, journalTitle=中国临床药理学杂志, columnName=综述, runingTitle=null, highlight=null, articleAbstract=
多囊卵巢综合征(PCOS)是育龄期女性高发的内分泌代谢紊乱性疾病,其中肥胖型PCOS以胰岛素抵抗(IR)和糖脂代谢异常为核心驱动因素。当前以二甲双胍联合胰高血糖素样肽-1受体激动剂(GLP-1RA)(如司美格鲁肽)为代表的西医疗法在减重和改善IR方面疗效确切,但面临胃肠道不良反应及停药易反弹等局限。中医学(TCM)认为该病以“肾虚为本,脾虚为枢,痰湿瘀血为标”,中医药在调控机体代谢与生殖内分泌方面具有多靶点优势。研究表明,中药复方联合针刺、穴位埋线等适宜技术与上述西药协同应用,不仅能通过协同调控腺苷酸活化蛋白激酶(AMPK)、磷脂酰肌醇3-激酶/蛋白激酶B(PI3K/Akt)等关键信号通路进一步强化减重与代谢改善疗效,还能有效优化卵巢微环境,提升子宫内膜容受性,并显著拮抗西药引发的胃肠道不良反应。本文系统梳理了二甲双胍、司美格鲁肽联合中医药治疗肥胖型PCOS的核心机制、循证证据及安全性,旨在为制定规范化的中西医整合治疗方案提供科学的理论依据,并为突破该病的临床长期管理瓶颈提供新思路。
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1, 邱丹花
1, 李华梅
1, 陈小丽
1, 王明媚
1, 李少云
1, 温预关
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黄春优(1983-),女,副主任中医师,主要从事妇产科疾病与药物评价方面的工作和研究
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