Article(id=1291704919849980133, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1291704771212234905, articleNumber=null, orderNo=null, doi=10.13699/j.cnki.1001-6821.2025.17.022, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=1745510400000, receivedDateStr=2025-04-25, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1785896051608, onlineDateStr=2026-08-05, pubDate=1758038400000, pubDateStr=2025-09-17, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1785896051608, onlineIssueDateStr=2026-08-05, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1785896051608, creator=13701087609, updateTime=1785896051608, updator=13701087609, issue=Issue{id=1291704771212234905, tenantId=1146029695717560320, journalId=1246415772164075586, year='2025', volume='41', issue='17', pageStart='2401', pageEnd='2550', issueExtLink='null', onlineDate='null', pubDate='1758038400000', pubDateStr='2025-09-17', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=null, createTime=1785896016147, creator='13701087609', updateTime=1785896900366, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1291708479866164175, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1291704771212234905, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1291708479866164176, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1291704771212234905, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=2526, endPage=2533, ext={EN=ArticleExt(id=1291704920034529510, articleId=1291704919849980133, tenantId=1146029695717560320, journalId=1246415772164075586, language=EN, title=Research progress on the regulation of mitochondrial dysfunction by traditional Chinese medicine in the intervention of Parkinson’s disease, columnId=1246531408630534361, journalTitle=Chinese Journal of Clinical Pharmacology, columnName=Review, runingTitle=null, highlight=null, articleAbstract=

With China’s transition into an aging society, Parkinson’s disease (PD) has emerged as the second most prevalent neurodegenerative disorder threatening the health of the elderly population. However, there is still a lack of ideal clinical drugs that can effectively treat PD or halt its progression. Mitochondrial dysfunction is one of the key pathogenic mechanisms underlying PD, involving abnormalities in energy metabolism, mitophagy, oxidative stress, calcium homeostasis, mitochondrial dynamics and apoptosis. In recent years, traditional Chinese medicine (TCM) has demonstrated promising efficacy in treating PD by targeting mitochondrial function. Based on the critical role of mitochondrial dysfunction in PD pathogenesis, this article focuses on the research progress of TCM monomers and compound formulations in intervening PD by regulating pathways related to mitochondrial dysfunction. The aim is to provide a theoretical foundation and novel insights for further research into TCM-based mitochondrial-targeted therapies for PD.

, authors=Jian-qing LIANG^{1, 2}, Zi-wei Lܳ, Jian-cheng HE⁴, authorsList=Jian-qing LIANG, Zi-wei LÜ, Jian-cheng HE, authorCompany=null, correspAuthors=Jian-qing LIANG, authorNote=null, correspAuthorsNote=null, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1291704920462348519, articleId=1291704919849980133, tenantId=1146029695717560320, journalId=1246415772164075586, language=CN, title=中医药调控线粒体功能障碍干预帕金森病的研究进展, columnId=1246531408819278046, journalTitle=中国临床药理学杂志, columnName=综述, runingTitle=null, highlight=null, articleAbstract=

我国进入老龄化社会的当下，帕金森病（PD）成为危害老年人群健康的第2大神经退行性疾病，而临床尚缺乏能够有效治疗PD或阻止其病情进展的理想药物。线粒体功能障碍是PD的关键致病机制之一，其涉及能量代谢、线粒体自噬、氧化应激、钙稳态、线粒体动力学、细胞凋亡等途径异常。近年来，通过中药干预线粒体功能治疗PD取得良好疗效，本文基于线粒体功能障碍在PD发病中的关键作用，重点阐述中药单体、中药复方通过调控线粒体功能障碍相关途径干预PD的研究进展，以期为中药靶向线粒体治疗PD的深入研究提供理论依据和新的思路。

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梁建庆（1976-），男，教授/副主任医师，主要从事中医药防治帕金森病和肿瘤方面的临床工作和研究

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梁建庆（1976-），男，教授/副主任医师，主要从事中医药防治帕金森病和肿瘤方面的临床工作和研究

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