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肌少-骨质疏松症是由骨质疏松症与肌少症共存所致的衰老相关综合征，已严重威胁到老年人群身体健康。丝裂原活化蛋白激酶（MAPKs）信号通路，包括细胞外信号调节激酶1/2（ERK1/2）、p38 MAPK和c-Jun氨基末端激酶（JNK）等，可调控骨骼肌及骨组织的增殖、分化及凋亡，并在肌少-骨质疏松症的发生发展中扮演重要角色。近年来，多种中药（TCM）单体在调控MAPKs信号通路、影响成骨分化或肌肉萎缩相关过程方面显示出一定实验研究价值。本文就MAPKs信号通路在成骨-成肌中的调控效应作一综述，并总结了多种中药单体通过该通路干预肌少-骨质疏松症的研究进展，旨在为肌少-骨质疏松症的防治提供新的理论依据和治疗策略。

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