Article(id=1292135462555116520, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292135425250976333, articleNumber=null, orderNo=null, doi=10.13699/j.cnki.1001-6821.2026.10.017, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=1777564800000, receivedDateStr=2026-05-01, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1785998700988, onlineDateStr=2026-08-06, pubDate=1779897600000, pubDateStr=2026-05-28, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1785998700988, onlineIssueDateStr=2026-08-06, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1785998700988, creator=13701087609, updateTime=1785998700988, updator=13701087609, issue=Issue{id=1292135425250976333, tenantId=1146029695717560320, journalId=1246415772164075586, year='2026', volume='42', issue='10', pageStart='1351', pageEnd='1500', issueExtLink='null', onlineDate='null', pubDate='1779897600000', pubDateStr='2026-05-28', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=null, createTime=1785998692095, creator='13701087609', updateTime=1786014373634, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1292201198489202995, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292135425250976333, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1292201198489202996, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292135425250976333, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=1457, endPage=1461, ext={EN=ArticleExt(id=1292135462806774761, articleId=1292135462555116520, tenantId=1146029695717560320, journalId=1246415772164075586, language=EN, title=Long-acting C-type natriuretic peptide prodrug Navepegritide, a new approveddrug for the treatment of achondroplasia, columnId=1291339032068354347, journalTitle=Chinese Journal of Clinical Pharmacology, columnName=New Drugs Introduction, runingTitle=null, highlight=null, articleAbstract=

Achondroplasia (ACH) is the most common hereditary short limb dwarfism, caused by functional acquired mutations in the fibroblast growth factor receptor 3 (FGFR3) gene. In recent years, significant breakthroughs have been made in precision therapy targeting downstream pathways of FGFR3. Navepegritide (product name: YUVIWEL) is a long-acting C-type natriuretic peptide (CNP) prodrug that uses transient conjugation (TransCon) technology to couple CNP to PEG carrier molecules through a cleavable linker, achieving weekly subcutaneous administration. It was approved by the US Food and Drug Administration (FDA) in February 2026 to promote linear growth in children aged 2 years and above with non-closed epiphyseal plates and cartilage dysplasia. This article introduces the pharmacological properties, key clinical trial evidence, safety features, and clinical value of navepegritide, providing reference for clinical medication.

, authors=Yan LI¹, Xin HE², Shuang HU², authorsList=Yan LI, Xin HE, Shuang HU, authorCompany=null, correspAuthors=Shuang HU, authorNote=null, correspAuthorsNote=null, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1292135463184262122, articleId=1292135462555116520, tenantId=1146029695717560320, journalId=1246415772164075586, language=CN, title=治疗软骨发育不全的新药—长效C型利钠肽前药Navepegritide, columnId=1291339032219349293, journalTitle=中国临床药理学杂志, columnName=新药介绍, runingTitle=null, highlight=null, articleAbstract=

软骨发育不全（ACH）是一种常见的遗传性侏儒症，由成纤维细胞生长因子受体3（FGFR3）基因功能获得性突变导致。近年来，靶向FGFR3下游通路的精准治疗取得重大突破。Ascendis Pharma A/S公司研发的Navepegritide（商品名：YUVIWEL）是一种长效C型利钠肽（CNP）前药，采用瞬时偶联（TransCon）技术将CNP-38与惰性聚乙二醇载体连接，实现每周1次皮下给药并维持持续暴露，于2026年2月获得美国食品药品监督管理局（FDA）加速批准，用于促进2岁及以上骨骺未闭合的软骨发育不全患儿的线性生长。本文介绍Navepegritide的药理学特性、关键临床试验证据、安全性特征及其临床价值，为临床用药提供参考。

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李妍（1991-），女，主管药师，主要从事临床药学工作

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李妍（1991-），女，主管药师，主要从事临床药学工作

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