Article(id=1292126391441576812, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292126355785802497, articleNumber=null, orderNo=null, doi=10.13699/j.cnki.1001-6821.2026.04.015, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=1770825600000, receivedDateStr=2026-02-12, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1785996538266, onlineDateStr=2026-08-06, pubDate=1772208000000, pubDateStr=2026-02-28, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1785996538266, onlineIssueDateStr=2026-08-06, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1785996538266, creator=13701087609, updateTime=1785996538266, updator=13701087609, issue=Issue{id=1292126355785802497, tenantId=1146029695717560320, journalId=1246415772164075586, year='2026', volume='42', issue='4', pageStart='451', pageEnd='600', issueExtLink='null', onlineDate='null', pubDate='1772208000000', pubDateStr='2026-02-28', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=null, createTime=1785996529766, creator='13701087609', updateTime=1786014519902, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1292201811952292026, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292126355785802497, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1292201811952292027, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292126355785802497, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=548, endPage=551, ext={EN=ArticleExt(id=1292126393257710445, articleId=1292126391441576812, tenantId=1146029695717560320, journalId=1246415772164075586, language=EN, title=A novel drug for treating thrombotic microangiopathy associated with hematopoietic stem cell transplantation-Narsoplimab, columnId=1291339032068354347, journalTitle=Chinese Journal of Clinical Pharmacology, columnName=New Drugs Introduction, runingTitle=null, highlight=null, articleAbstract=

Narsoplimab was first approved by the U.S. Food and Drug Administration (FDA) on December 24,2025, for the management of thrombotic microangiopathy following hematopoietic stem cell transplantation (TA-TMA) in children aged 2 years and older and adults. As a serious complication of hematopoietic stem cell transplantation (HSCT), TA-TMA is characterized by high mortality. TA-TMA is associated with endothelial injury and can activate the complement agglutinin pathway, where mannose-binding agglutinin-related serine protease-2 (MASP-2) is a key enzyme. Narsoplimab is a humanized monoclonal antibody that specifically binds to and inhibits MASP-2, thereby reducing complement activation in damaged tissues without compromising the beneficial role of complement in anti-infection. Multiple real-world studies have demonstrated a high response rate. This article provides an overview of the mechanism of action, pharmacodynamics, pharmacokinetics, clinical studies, and safety profile of Narsoplimab.

, authors=Ting-ting RUAN¹, Yan-yan JIA¹, Hua-qing DU¹, Ting-ting BIAN², authorsList=Ting-ting RUAN, Yan-yan JIA, Hua-qing DU, Ting-ting BIAN, authorCompany=null, correspAuthors=Ting-ting BIAN, authorNote=null, correspAuthorsNote=null, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1292126393513562990, articleId=1292126391441576812, tenantId=1146029695717560320, journalId=1246415772164075586, language=CN, title=治疗造血干细胞移植相关血栓性微血管病的新药Narsoplimab, columnId=1291339032219349293, journalTitle=中国临床药理学杂志, columnName=新药介绍, runingTitle=null, highlight=null, articleAbstract=

Narsoplimab于2025年12月24日由美国食品药品监督管理局批准，成为首个用于治疗2岁及以上儿童与成人造血干细胞移植相关血栓性微血管病（TA-TMA）的药物。TA-TMA是造血干细胞移植（HSCT）后较为常见且病情凶险的一类并发症，具有较高死亡率。TA-TMA与内皮损伤有关，可激活补体凝集素途径，甘露聚糖集合凝集素相关丝氨酸蛋白酶-2（MASP-2）是该途径中的关键酶。Narsoplimab是一种人源单克隆抗体，能够特异性结合并抑制MASP-2，可降低受损组织中的补体激活，同时不破坏补体在抗感染中的有益作用，在多项真实世界研究中显示出了较高的缓解率。现对Narsoplimab的作用机制、药效学特征、药代动力学特点、临床研究进展及安全性研究现状等做一分析与介绍。

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阮婷婷（1987-），女，主管药师，主要从事妇科药学相关工作与研究

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阮婷婷（1987-），女，主管药师，主要从事妇科药学相关工作与研究

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