Article(id=1292126566532805268, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292126460546929080, articleNumber=null, orderNo=null, doi=10.13699/j.cnki.1001-6821.2026.03.017, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=1751558400000, receivedDateStr=2025-07-04, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1785996580011, onlineDateStr=2026-08-06, pubDate=1771257600000, pubDateStr=2026-02-17, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1785996580011, onlineIssueDateStr=2026-08-06, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1785996580011, creator=13701087609, updateTime=1785996580011, updator=13701087609, issue=Issue{id=1292126460546929080, tenantId=1146029695717560320, journalId=1246415772164075586, year='2026', volume='42', issue='3', pageStart='301', pageEnd='450', issueExtLink='null', onlineDate='null', pubDate='1771257600000', pubDateStr='2026-02-17', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=null, createTime=1785996554742, creator='13701087609', updateTime=1786014541627, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1292201903060963536, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292126460546929080, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1292201903060963537, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292126460546929080, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=405, endPage=411, ext={EN=ArticleExt(id=1292126566750909077, articleId=1292126566532805268, tenantId=1146029695717560320, journalId=1246415772164075586, language=EN, title=A systematic review of the efficacy and safety of L-carnitine in the treatment of myocardial damage, columnId=1246531412438966969, journalTitle=Chinese Journal of Clinical Pharmacology, columnName=Reader’s Field, runingTitle=null, highlight=null, articleAbstract=
Objective To systematically evaluate the efficacy and safety of lecarnitine in the treatment of patients with myocardial damage.
Methods Search databases such as PubMed, Embase, Cochrane Library, CNKI, Wanfang Database, etc. randomized controlled trials (RCTS) comparing L-carnitine with sodium creatine phosphate, sodium fructobisphosphonate, trimetazidine and coenzyme Q10 for the treatment of myocardial damage were included according to the inclusion and exclusion criteria. The search period for all was from the establishment of the database to May 2024. Meta-analysis was performed using RevMan 5.3 and Stata 14.0.
Results A total of 12 RCTs were included, involving a total of 1 408 patients. Meta-analysis showed that the total effective rate of levocarnitine was 89.53% (556 cases/621 cases), which was significantly higher than that [83.98% (519 cases/618 cases)]of the control group [OR=1.83, 95% CI (1.10~3.06), P<0.05]; the incidence of adverse drug reactions of levocarnitine and the control group was 2.47% (9 cases/364 cases) and 3.31% (12 cases/362 cases) with no statistically significant difference [OR=0.83, 95% CI (0.17~4.18), P>0.05]. The creatine kinase (CK) levels of L-carnitine group and the control group were(203.04±34.59) and (238.28±33.97)U·L-1, respectvely [OR=-26.81, 95% CI (-47.43~-6.18), P<0.01]; the creatine kinase isoenzyme MB (CK-MB) levels were (21.64±10.76) and (26.71±14.48)U·L-1, respectvely [OR=-4.19, 95% CI (-6.70~-1.67), P<0.001]were significantly better than those of . There was no statistically significant difference in the levels of lactate dehydrogenase (LDH) and cardiac troponin Ⅰ (cTnⅠ) of L-carnitine compared with the control group (all P>0.05).
Conclusion The effectiveness of levocarnitine in the treatment of myocardial damage is relatively good, and there are no obvious adverse reactions.
, authors=Jun-xian LIN
1, 4, Zhuo CHEN
2, Qi-qi WU
2, Lu LIU
3, Mei-xing YAN
3, authorsList=Jun-xian LIN, Zhuo CHEN, Qi-qi WU, Lu LIU, Mei-xing YAN, authorCompany=null, correspAuthors=Mei-xing YAN, authorNote=null, correspAuthorsNote=null, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1292126567023538838, articleId=1292126566532805268, tenantId=1146029695717560320, journalId=1246415772164075586, language=CN, title=左卡尼汀治疗心肌损害有效性和安全性的系统评价, columnId=1246531414020219657, journalTitle=中国临床药理学杂志, columnName=读者园地, runingTitle=null, highlight=null, articleAbstract=
目的 系统评价左卡尼汀治疗心肌损害患者的有效性和安全性。
方法 检索PubMed、Embase、Cochrane Library、中国知网、万方数据库等数据库，根据纳排标准纳入左卡尼汀对比磷酸肌酸钠、果糖二膦酸钠、曲美他嗪以及辅酶Q10治疗心肌损害的随机对照试验（RCT），检索时限均为建库起至2024年5月。用RevMan 5.3和Stata 14.0进行Meta分析。
结果 共纳入12项RCT，共计1 408例患者。Meta分析显示，左卡尼汀患者组和其他药物组的的总有效率分别为89.53%（556例/621例）和83.98%（519例/618例），左卡尼汀患者组显著高于其他药物组[OR=1.83，95％CI（1.10~3.06），P<0.05]；左卡尼汀组和对照组的药物不良反应发生率与分别为2.47%（9例/364例）和3.31%（12例/36例），2组比较在统计学上差异无统计学意义[OR=0.83，95％CI（0.17~4.18），P>0.05]。左卡尼汀组和对照组的肌酸激酶（CK）水平分别为（203.04±34.59）和（238.28±33.97）U·L-1，2组间比较在统计学上差异有统计学意义[OR=-26.81，95％CI（-47.43~-6.18），P<0.01]，肌酸激酶同工酶MB（CK-MB）水平分别为（21.64±10.76）和（26.71±14.48）U·L-1，2组间比较在统计学上差异有统计学意义[OR=-4.19，95％CI（-6.70~-1.67），P<0.001]；左卡尼汀的乳酸脱氢酶（LDH）及心肌肌钙蛋白I（cTnI）水平与对照组相比，在统计学上差异均无统计学意义（均P>0.05）。
结论 左卡尼汀组患者的总有效率显著高于其他药物组，且无明显的药物不良反应。
, authors=林均贤
1, 4, 陈卓
2, 吴琪琪
2, 刘璐
3, 闫美兴
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林均贤（1993-），女，主管药师，主要从事临床药学的相关工作和研究
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