Article(id=1292126404464890845, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292126377440989952, articleNumber=null, orderNo=null, doi=10.13699/j.cnki.1001-6821.2026.01.021, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=1760976000000, receivedDateStr=2025-10-21, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1785996541372, onlineDateStr=2026-08-06, pubDate=1768579200000, pubDateStr=2026-01-17, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1785996541372, onlineIssueDateStr=2026-08-06, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1785996541372, creator=13701087609, updateTime=1785996541372, updator=13701087609, issue=Issue{id=1292126377440989952, tenantId=1146029695717560320, journalId=1246415772164075586, year='2026', volume='42', issue='1', pageStart='1', pageEnd='147', issueExtLink='null', onlineDate='null', pubDate='1768579200000', pubDateStr='2026-01-17', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=null, createTime=1785996534929, creator='13701087609', updateTime=1786014708731, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1292202603945947673, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292126377440989952, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1292202603950141978, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1292126377440989952, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=131, endPage=136, ext={EN=ArticleExt(id=1292126404670411742, articleId=1292126404464890845, tenantId=1146029695717560320, journalId=1246415772164075586, language=EN, title=Research status on the mechanisms of traditional Chinese medicine treatment for ischemic stroke based on network pharmacology and metabolomics, columnId=1246531408630534361, journalTitle=Chinese Journal of Clinical Pharmacology, columnName=Review, runingTitle=null, highlight=null, articleAbstract=

Ischemic stroke, characterized by reduced cerebral blood flow leading to neurological dysfunction, has high mortality and disability rates. Traditional Chinese medicine (TCM) not only alleviates symptoms but also reduces complications, making it widely used clinically. However, due to the multi-component, multi-target and multi-pathway nature of TCM, the mechanisms of many drugs remain unclear. Network pharmacology and metabolomics, as branches of systems biology, have become hotspots in TCM research. Network pharmacology reveals drug mechanisms through target network analysis, while metabolomics assesses metabolic pathway alterations. This review analyzes recent literature from three aspects, including single herbs, herb pairs and compound prescriptions, summarizing advances in network pharmacology and metabolomics research on TCM for ischemic stroke, providing a systematic theoretical basis for clinical application.

, authors=Pei-hua LIANG¹, Ke-jiang GAO², LÜ-xu WANG¹, Yong-li LIU¹, Yu-zhen LI¹, authorsList=Pei-hua LIANG, Ke-jiang GAO, LÜ-xu WANG, Yong-li LIU, Yu-zhen LI, authorCompany=null, correspAuthors=Yu-zhen LI, authorNote=null, correspAuthorsNote=null, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1292126404913681375, articleId=1292126404464890845, tenantId=1146029695717560320, journalId=1246415772164075586, language=CN, title=网络药理学与代谢组学揭示中药治疗缺血性脑卒中作用机制研究现状, columnId=1246531408819278046, journalTitle=中国临床药理学杂志, columnName=综述, runingTitle=null, highlight=null, articleAbstract=

缺血性脑卒中是由于脑部血流减少导致大脑功能异常的疾病，具有高致死和致残率。中医药（TCM）治疗缺血性脑卒中不仅能够改善症状，还能减少并发症，在临床上应用广泛。由于中药治疗“多成分-多靶点-多途径”的特点，许多药物的作用机制尚不明确。网络药理学和代谢组学作为系统生物学的分支，成为近年来中药研究领域的热点，网络药理学通过对药物靶点的网络分析，揭示药物作用机制，而代谢组学则能够评估药物对代谢途径的影响。本文通过对近年来相关领域的文献进行分析，从单味中药、配伍药对及中药复方3个方面，总结了基于网络药理学和代谢组学研究中药治疗缺血性脑卒中的研究进展，为中药治疗缺血性脑卒中的临床应用提供更加系统的理论依据。

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梁佩华（1995-），女，主管药师，主要从事临床药学方面的工作和研究

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梁佩华（1995-），女，主管药师，主要从事临床药学方面的工作和研究

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