Article(id=1291707127131824770, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1291707004800750583, articleNumber=null, orderNo=null, doi=10.13699/j.cnki.1001-6821.2025.22.014, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=1753977600000, receivedDateStr=2025-08-01, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1785896577865, onlineDateStr=2026-08-05, pubDate=1764259200000, pubDateStr=2025-11-28, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1785896577865, onlineIssueDateStr=2026-08-05, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1785896577865, creator=13701087609, updateTime=1785896577865, updator=13701087609, issue=Issue{id=1291707004800750583, tenantId=1146029695717560320, journalId=1246415772164075586, year='2025', volume='41', issue='22', pageStart='3151', pageEnd='3300', issueExtLink='null', onlineDate='null', pubDate='1764259200000', pubDateStr='2025-11-28', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=null, createTime=1785896548700, creator='13701087609', updateTime=1785896693213, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1291707610999316815, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1291707004800750583, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1291707610999316816, tenantId=1146029695717560320, journalId=1246415772164075586, issueId=1291707004800750583, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=3241, endPage=3247, ext={EN=ArticleExt(id=1291707127517700739, articleId=1291707127131824770, tenantId=1146029695717560320, journalId=1246415772164075586, language=EN, title=Research status of traditional Chinese medicine on mitochondrial quality control in the treatment of cardiac fibrosis, columnId=1246531408630534361, journalTitle=Chinese Journal of Clinical Pharmacology, columnName=Review, runingTitle=null, highlight=null, articleAbstract=

Cardiac fibrosis is a common pathological basis for a variety of cardiovascular diseases, and its core feature is cardiac fibroblast overactivation and extracellular matrix deposition, which ultimately leads to cardiac structural remodeling. Studies have shown that mitochondrial dysfunction is an important driver of the occurrence and development of cardiac fibrosis. Mitochondrial quality control is a set of precise dynamic regulatory systems in cells, involving mitochondrial dynamics, mitochondrial biogenesis and mitochondrial autophagy, which are responsible for maintaining mitochondrial homeostasis. Traditional Chinese medicine has the advantages of multiple pathways, multiple targets and small toxic side effects, and shows great potential in regulating mitochondrial quality control and treating cardiac fibrosis. Based on this, this paper systematically reviews the relationship between mitochondrial quality control and cardiac fibrosis and the mechanism of traditional Chinese medicine to regulate mitochondrial quality control in the treatment of cardiac fibrosis, aiming to provide a theoretical basis and new targets for the clinical treatment of cardiovascular diseases.

, authors=Jie LIU, Hua JIN, Jie DU, Xuan BAO, authorsList=Jie LIU, Hua JIN, Jie DU, Xuan BAO, authorCompany=null, correspAuthors=Hua JIN, authorNote=null, correspAuthorsNote=null, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1291707127983268484, articleId=1291707127131824770, tenantId=1146029695717560320, journalId=1246415772164075586, language=CN, title=中医药调控线粒体质量控制治疗心脏纤维化的研究现状, columnId=1246531408819278046, journalTitle=中国临床药理学杂志, columnName=综述, runingTitle=null, highlight=null, articleAbstract=

心脏纤维化是多种心血管疾病的共同病理基础，其核心特征是心脏成纤维细胞过度活化与细胞外基质沉积，最终导致心脏结构重塑。研究显示，线粒体功能障碍是心脏纤维化发生发展的重要驱动因素。线粒体质量控制是细胞内一套精密的动态调控系统，涉及线粒体动力学、线粒体生物发生和线粒体自噬等过程，负责维持线粒体稳态，故线粒体质量控制紊乱介导心脏纤维化病理进程。中医药具有多途径、多靶点和毒副作用小等优势，在调控线粒体质量控制和治疗心脏纤维化方面展现出巨大潜力。基于此，本文就线粒体质量控制与心脏纤维化的关系以及中医药调控线粒体质量控制治疗心脏纤维化的作用机制展开系统综述，旨在为临床治疗心血管疾病提供理论依据与新靶点。

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刘杰（1999-），女，硕士研究生，从事中医药防治心脑血管疾病的研究

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刘杰（1999-），女，硕士研究生，从事中医药防治心脑血管疾病的研究

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