Article(id=1211269160370311415, tenantId=1146029695717560320, journalId=1189873630562394117, issueId=1211269157790806494, articleNumber=null, orderNo=null, doi=10.11855/j.issn.0577-7402.2021.04.15, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=research-article, receivedDate=1609171200000, receivedDateStr=2020-12-29, revisedDate=1616083200000, revisedDateStr=2021-03-19, acceptedDate=null, acceptedDateStr=null, onlineDate=1766718672125, onlineDateStr=2025-12-26, pubDate=1619539200000, pubDateStr=2021-04-28, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1766718672125, onlineIssueDateStr=2025-12-26, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1766718672125, creator=13701087609, updateTime=1766718672125, updator=13701087609, issue=Issue{id=1211269157790806494, tenantId=1146029695717560320, journalId=1189873630562394117, year='2021', volume='46', issue='4', pageStart='319', pageEnd='424', issueExtLink='null', onlineDate='null', pubDate='null', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=0, createTime=1766718671510, creator=13701087609, updateTime=1766718756000, updator=13701087609, preIssue=null, nextIssue=null, ext={EN=IssueExt(id=1211269512217882745, tenantId=1146029695717560320, journalId=1189873630562394117, issueId=1211269157790806494, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1211269512217882746, tenantId=1146029695717560320, journalId=1189873630562394117, issueId=1211269157790806494, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null}, startPage=410, endPage=413, ext={EN=ArticleExt(id=1211269160718438663, articleId=1211269160370311415, tenantId=1146029695717560320, journalId=1189873630562394117, language=EN, title=Role of C-type lectin-like receptor 2 in thrombosis, columnId=1190243275882729994, journalTitle=Medical Journal of Chinese People’s Liberation Army, columnName=Review, runingTitle=null, highlight=null, articleAbstract=

C-type lectin-like receptor 2 (CLEC-2) is a member of the cell surface receptor C-type lectin superfamily. CLEC-2 expressed in platelet surface is a platelet activatory receptor based on the immune receptor tyrosine activation motif, which can participate in platelet activation and aggregation by binding to its ligand. This physiological process can prevent excessive blood loss in the body, but is also the pathological basis of many thromboembolic diseases. CLEC-2 and its ligands are involved in the pathophysiological processes such as atherosclerosis, inflammation thrombotic state, maintenance of vascular integrity, and cancer-related thrombosis. The important role of CLEC-2 in the thrombosis process has been reviewed in present paper from the four aspects mentioned above.

, correspAuthors=Xing-Shun Qi, authorNote=null, correspAuthorsNote=, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, authorCompany=null, fund=null, authors=null, authorsList=Shi-Xue Xu, Yang Yang, Yang-Lan He, Xing-Shun Qi), CN=ArticleExt(id=1211269162769453362, articleId=1211269160370311415, tenantId=1146029695717560320, journalId=1189873630562394117, language=CN, title=C型凝集素样受体-2在血栓形成中的作用研究进展, columnId=1190243276029530637, journalTitle=解放军医学杂志, columnName=综述, runingTitle=null, highlight=null, articleAbstract=

C型凝集素样受体-2(CLEC-2)是细胞表面受体C型凝集素超家族的一员。表达于血小板表面的CLEC-2是一种基于免疫受体酪氨酸活化基序的血小板活化受体，可通过与其配体结合参与血小板的活化及聚集。这一生理过程可以避免机体过度失血，但同时也是多种血栓栓塞性疾病的病理基础。在动脉粥样硬化、炎症血栓状态、血管壁完整性的维持、癌症相关血栓形成等病理生理过程中均有CLEC-2及其配体的参与。该文从以上4个方面对CLEC-2在血栓形成过程中的重要作用进行综述。

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徐士雪，硕士研究生，主要从事消化系统疾病诊治方面的研究

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CLEC-2. C型凝集素样受体-2；syk. 脾酪氨酸激酶；SLP-76. 含有SH2结构域的76 ku的白细胞蛋白；PLCγ2. 磷脂酶γ2

, figureFileSmall=wiF8SQiAbbK5VYBpXO8GMg==, figureFileBig=63sismj8NiHVAXPDbMMU9Q==, tableContent=null), ArticleFig(id=1211269167123141154, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1211269160370311415, language=EN, label=Fig.2, caption=Major pathways of CLEC-2 mediating thrombosis, figureFileSmall=gqj7WNieutFKUybQc6M05Q==, figureFileBig=KeXoEQDtEn4eroauBxMwkg==, tableContent=null), ArticleFig(id=1211269167232193064, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1211269160370311415, language=CN, label=图2, caption=CLEC-2介导血栓形成的主要途径

CLEC-2. C型凝集素样受体-2；PDPN. 平足蛋白；S100A13.S100钙结合蛋白A13；ITAM. 免疫受体酪氨酸活化基序

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