Article(id=1208862369971826940, tenantId=1146029695717560320, journalId=1189873630562394117, issueId=1208862365714616539, articleNumber=null, orderNo=null, doi=10.11855/j.issn.0577-7402.2021.09.04, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=research-article, receivedDate=1619366400000, receivedDateStr=2021-04-26, revisedDate=1625500800000, revisedDateStr=2021-07-06, acceptedDate=null, acceptedDateStr=null, onlineDate=1766144848577, onlineDateStr=2025-12-19, pubDate=1632758400000, pubDateStr=2021-09-28, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1766144848577, onlineIssueDateStr=2025-12-19, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1766144848577, creator=13701087609, updateTime=1766144848577, updator=13701087609, issue=Issue{id=1208862365714616539, tenantId=1146029695717560320, journalId=1189873630562394117, year='2021', volume='46', issue='9', pageStart='849', pageEnd='953', issueExtLink='null', onlineDate='null', pubDate='null', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=0, createTime=1766144847562, creator=13701087609, updateTime=1766144914151, updator=13701087609, preIssue=null, nextIssue=null, ext={EN=IssueExt(id=1208862645055254758, tenantId=1146029695717560320, journalId=1189873630562394117, issueId=1208862365714616539, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1208862645055254759, tenantId=1146029695717560320, journalId=1189873630562394117, issueId=1208862365714616539, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null}, startPage=865, endPage=870, ext={EN=ArticleExt(id=1208862370403840267, articleId=1208862369971826940, tenantId=1146029695717560320, journalId=1189873630562394117, language=EN, title=Research progress on the relationship of intestinal flora-mediated immune response to hypertension and chronic kidney disease, columnId=1208862370294788358, journalTitle=Medical Journal of Chinese People’s Liberation Army, columnName=Special Issues on Intestinal Flora and Disease Progression, runingTitle=null, highlight=null, articleAbstract=

Intestinal flora is increasingly considered to be associated with multiple diseases, such as insulin resistance, bile acid metabolism and inflammatory reaction. Research on the relationship of intestinal flora to the occurrence and development of hypertension and chronic kidney disease has made remarkable progress in recent years. The occurrence and development of hypertension and chronic kidney disease disrupt the original gut microbiota composition, further contribute to the advancement of hypertension and chronic kidney disease. So far, further discussion is needed on the causality and exact mechanism of intestinal flora to hypertension and chronic kidney disease. The composition of intestinal microorganisms in hypertension and chronic kidney disease has been reviewed in present paper, and focused on the role of symbiotic microorganisms in affecting the host immunity and the progress of the primary disease, and summarized the relationship of intestinal flora mediated immune response to hypertension and chronic kidney disease, mainly interlinked each other through short-chain fatty acids, angiotensin Ⅱ and trimethylamine oxide to provide the basis for finding potential treatment methods for hypertension and chronic kidney disease.

, correspAuthors=Hong-Ying Peng, authorNote=null, correspAuthorsNote=, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, authorCompany=null, fund=null, authors=null, authorsList=Jing Yu, Hong-Ying Peng), CN=ArticleExt(id=1208862370881990943, articleId=1208862369971826940, tenantId=1146029695717560320, journalId=1189873630562394117, language=CN, title=肠道菌群介导的免疫反应与高血压和慢性肾脏病的关系研究进展, columnId=1208862370521280782, journalTitle=解放军医学杂志, columnName=肠道菌群与疾病的关系研究进展专题, runingTitle=null, highlight=null, articleAbstract=

肠道菌群被认为与多种疾病相关，其中，肠道菌群与高血压、慢性肾脏病发生及发展的相关性研究近年来取得了较大进展。高血压和慢性肾脏病的发生、发展破坏了原有的肠道菌群组成，进而又促进了高血压和慢性肾脏病的发展。高血压、慢性肾脏病与肠道菌群改变的因果关系及其确切机制仍需进一步探讨。该文回顾了高血压和慢性肾脏病患者的肠道微生物组成，重点总结了共生微生物影响宿主免疫对原发疾病进展的作用，阐述了肠道菌群介导的免疫反应与高血压、慢性肾脏病之间除细菌细胞壁及脂多糖外主要通过短链脂肪酸、血管紧张素Ⅱ、氧化三甲胺等相互联系，以期为寻找治疗高血压和慢性肾脏疾病的潜在方法提供参考。

, correspAuthors=彭红英, authorNote=null, correspAuthorsNote=, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=IyDJBfnXOCV+aI+bjB0vyQ==, magXml=FWbjiFTtSmAIe7ypQslNEw==, pdfUrl=null, pdf=+P6NuNBSPtkZ2xc3euCMpQ==, pdfFileSize=816175, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=dh1DkjRfnWTWu5EUozso9w==, mapNumber=null, authorCompany=null, fund=null, authors=, authorsList=喻静, 彭红英)}, authors=[Author(id=1208862371410473277, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, orderNo=0, firstName=null, middleName=null, lastName=null, nameCn=null, orcid=null, stid=null, country=null, authorPic=null, dead=0, email=null, emailSecond=null, emailThird=null, correspondingAuthor=0, authorType=1, ext={EN=AuthorExt(id=1208862371498553664, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, authorId=1208862371410473277, language=EN, stringName=Jing Yu, firstName=Jing, middleName=null, lastName=Yu, prefix=null, suffix=null, authorComment=null, nameInitials=null, affiliation=null, department=null, xref=¹, address=¹Department of Nephrology, Affiliated Hospital of Guizhou Medical University, Guiyang 550004, China, bio=null, bioImg=null, bioContent=null, aboutCorrespAuthor=null), CN=AuthorExt(id=1208862371590828357, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, authorId=1208862371410473277, language=CN, stringName=喻静, firstName=静, middleName=null, lastName=喻, prefix=null, suffix=null, authorComment=null, nameInitials=null, affiliation=null, department=null, xref=¹, address=¹贵州医科大学附属医院肾内科，贵阳 550004, bio={"content":"

喻静，硕士研究生，住院医师，主要从事肾实质疾病方面的研究

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喻静，硕士研究生，住院医师，主要从事肾实质疾病方面的研究

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Microbiome and hypertension: where are we now?[J]. J Cardiovasc Med(Hagerstown), 2020, 21(2): 83-88., articleTitle=Microbiome and hypertension: where are we now?, refAbstract=null), Reference(id=1208862374380040626, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2021, volume=78, issue=3, pageStart=909, pageEnd=922, url=null, language=null, rfNumber=[2], rfOrder=1, authorNames=Liu JR, Miao H, Deng DQ, journalName=Cell Mol Life Sci, refType=null, unstructuredReference=Liu JR, Miao H, Deng DQ, et al. Gut microbiota-derived tryptophan metabolism mediates renal fibrosis by aryl hydrocarbon receptor signaling activation[J]. Cell Mol Life Sci, 2021, 78(3): 909-922., articleTitle=Gut microbiota-derived tryptophan metabolism mediates renal fibrosis by aryl hydrocarbon receptor signaling activation, refAbstract=null), Reference(id=1208862374455538104, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2019, volume=26, issue=2, pageStart=122, pageEnd=130, url=null, language=null, rfNumber=[3], rfOrder=2, authorNames=Barrows IR, Ramezani A, Raj DS, journalName=Adv Chronic Kidney Dis, refType=null, unstructuredReference=Barrows IR, Ramezani A, Raj DS. Inflammation, immunity, and oxidative stress in hypertension-partners in crime?[J]. Adv Chronic Kidney Dis, 2019, 26(2): 122-130., articleTitle=Inflammation, immunity, and oxidative stress in hypertension-partners in crime?, refAbstract=null), Reference(id=1208862374543618493, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2019, volume=10, issue=null, pageStart=2100, pageEnd=null, url=null, language=null, rfNumber=[4], rfOrder=3, authorNames=Murdoch CC, Rawls JF, journalName=Front Immunol, refType=null, unstructuredReference=Murdoch CC, Rawls JF. Commensal microbiota regulate vertebrate innate immunity-insights from the zebrafish[J]. Front Immunol, 2019, 10: 2100., articleTitle=Commensal microbiota regulate vertebrate innate immunity-insights from the zebrafish, refAbstract=null), Reference(id=1208862374669447618, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2018, volume=10, issue=3, pageStart=163, pageEnd=171, url=null, language=null, rfNumber=[5], rfOrder=4, authorNames=Heiss CN, Olofsson LE, journalName=J Innate Immun, refType=null, unstructuredReference=Heiss CN, Olofsson LE. Gut microbiota-dependent modulation of energy metabolism[J]. J Innate Immun, 2018, 10(3): 163-171., articleTitle=Gut microbiota-dependent modulation of energy metabolism, refAbstract=null), Reference(id=1208862374799471047, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2016, volume=5, issue=9, pageStart=771, pageEnd=781, url=null, language=null, rfNumber=[6], rfOrder=5, authorNames=Burcelin R, journalName=Mol Metab, refType=null, unstructuredReference=Burcelin R. Gut microbiota and immune crosstalk in metablic disease[J]. Mol Metab, 2016, 5(9): 771-781., articleTitle=Gut microbiota and immune crosstalk in metablic disease, refAbstract=null), Reference(id=1208862374879162831, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2012, volume=4, issue=137, pageStart=137rv7, pageEnd=null, url=null, language=null, rfNumber=[7], rfOrder=6, authorNames=Blumberg R, Powrie F, journalName=Sci Transl Med, refType=null, unstructuredReference=Blumberg R, Powrie F. Microbiota, disease, and back to health: a metastable journey[J]. Sci Transl Med, 2012, 4(137): 137rv7., articleTitle=Microbiota, disease, and back to health: a metastable journey, refAbstract=null), Reference(id=1208862374984020435, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2016, volume=23, issue=3, pageStart=413, pageEnd=426, url=null, language=null, rfNumber=[8], rfOrder=7, authorNames=Winer DA, Luck H, Tsai S, journalName=Cell Metab, refType=null, unstructuredReference=Winer DA, Luck H, Tsai S, et al. The intestinal immune system in obesity and insulin resistance[J]. Cell Metab, 2016, 23(3): 413-426., articleTitle=The intestinal immune system in obesity and insulin resistance, refAbstract=null), Reference(id=1208862375114043866, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2019, volume=221, issue=2, pageStart=146.e1, pageEnd=146.e23, url=null, language=null, rfNumber=[9], rfOrder=8, authorNames=Seferovic MD, Pace RM, Carroll M, journalName=Am J Obstet Gynecol, refType=null, unstructuredReference=Seferovic MD, Pace RM, Carroll M, et al. Visualization of microbes by 16S in situ hybridization in term and preterm placentas without intraamniotic infection[J]. Am J Obstet Gynecol, 2019, 221(2): 146.e1-146.e23., articleTitle=Visualization of microbes by 16S in situ hybridization in term and preterm placentas without intraamniotic infection, refAbstract=null), Reference(id=1208862375231484386, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2021, volume=2021, issue=null, pageStart=6655414, pageEnd=null, url=null, language=null, rfNumber=[10], rfOrder=9, authorNames=Tan Q, Zeng Z, Xu F, journalName=Can J Infect Dis Med Microbiol, refType=null, unstructuredReference=Tan Q, Zeng Z, Xu F, et al. A comparative transcriptomic analysis of human placental trophoblasts in response to pathogenic and probiotic Enterococcus faecalis interaction[J]. Can J Infect Dis Med Microbiol, 2021, 2021: 6655414., articleTitle=A comparative transcriptomic analysis of human placental trophoblasts in response to pathogenic and probiotic Enterococcus faecalis interaction, refAbstract=null), Reference(id=1208862375348924905, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2014, volume=505, issue=7484, pageStart=559, pageEnd=563, url=null, language=null, rfNumber=[11], rfOrder=10, authorNames=David LA, Maurice CF, Carmody RN, journalName=Nature, refType=null, unstructuredReference=David LA, Maurice CF, Carmody RN, et al. Diet rapidly and reproducibly alters the human gut microbiome[J]. Nature, 2014, 505(7484): 559-563., articleTitle=Diet rapidly and reproducibly alters the human gut microbiome, refAbstract=null), Reference(id=1208862375445393901, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2017, volume=18, issue=1, pageStart=46, pageEnd=null, url=null, language=null, rfNumber=[12], rfOrder=11, authorNames=Gao B, Tu P, Bian X, journalName=BMC Pharmacol Toxicol, refType=null, unstructuredReference=Gao B, Tu P, Bian X, et al. Profound perturbation induced by triclosan exposure in mouse gut microbiome: a less resilient microbial community with elevated antibiotic and metal resistomes[J]. BMC Pharmacol Toxicol, 2017, 18(1): 46., articleTitle=Profound perturbation induced by triclosan exposure in mouse gut microbiome: a less resilient microbial community with elevated antibiotic and metal resistomes, refAbstract=null), Reference(id=1208862375550251507, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2013, volume=62, issue=11, pageStart=1591, pageEnd=1601, url=null, language=null, rfNumber=[13], rfOrder=12, authorNames=Pérez-Cobas AE, Gosalbes MJ, Friedrichs A, journalName=Gut, refType=null, unstructuredReference=Pérez-Cobas AE, Gosalbes MJ, Friedrichs A, et al. Gut microbiota disturbance during antibiotic therapy: a multi-omic approach[J]. Gut, 2013, 62(11): 1591-1601., articleTitle=Gut microbiota disturbance during antibiotic therapy: a multi-omic approach, refAbstract=null), Reference(id=1208862375634137592, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2018, volume=67, issue=4, pageStart=625, pageEnd=633, url=null, language=null, rfNumber=[14], rfOrder=13, authorNames=Barton W, Penney NC, Cronin O, journalName=Gut, refType=null, unstructuredReference=Barton W, Penney NC, Cronin O, et al. The microbiome of professional athletes differs from that of more sedentary subjects in composition and particularly at the functional metabolic level[J]. Gut, 2018, 67(4): 625-633., articleTitle=The microbiome of professional athletes differs from that of more sedentary subjects in composition and particularly at the functional metabolic level, refAbstract=null), Reference(id=1208862375738995196, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2019, volume=115, issue=9, pageStart=1435, pageEnd=1447, url=null, language=null, rfNumber=[15], rfOrder=14, authorNames=Jama HA, Beale A, Shihata WA, journalName=Cardiovasc Res, refType=null, unstructuredReference=Jama HA, Beale A, Shihata WA, et al. The effect of diet on hypertensive pathology: is there a link via gut microbiota-driven immunometabolism?[J]. Cardiovasc Res, 2019, 115(9): 1435-1447., articleTitle=The effect of diet on hypertensive pathology: is there a link via gut microbiota-driven immunometabolism?, refAbstract=null), Reference(id=1208862375860630016, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2015, volume=65, issue=6, pageStart=1331, pageEnd=1340, url=null, language=null, rfNumber=[16], rfOrder=15, authorNames=Yang T, Santisteban MM, Rodriguez V, journalName=Hypertension, refType=null, unstructuredReference=Yang T, Santisteban MM, Rodriguez V, et al. Gut dysbiosis is linked to hypertension[J]. Hypertension, 2015, 65(6): 1331-1340., articleTitle=Gut dysbiosis is linked to hypertension, refAbstract=null), Reference(id=1208862375957099012, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2019, volume=32, issue=1, pageStart=27, pageEnd=37, url=null, language=null, rfNumber=[17], rfOrder=16, authorNames=Cosola C, Rocchetti MT, Sabatino A, journalName=J Nephrol, refType=null, unstructuredReference=Cosola C, Rocchetti MT, Sabatino A, et al. Microbiota issue in CKD: how promising are gut-targeted approaches?[J]. J Nephrol, 2019, 32(1): 27-37., articleTitle=Microbiota issue in CKD: how promising are gut-targeted approaches?, refAbstract=null), Reference(id=1208862376020013578, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2021, volume=14, issue=null, pageStart=2177, pageEnd=2188, url=null, language=null, rfNumber=[18], rfOrder=17, authorNames=Zhou Q, Pang G, Zhang Z, journalName=Diabetes Metab Syndr Obes, refType=null, unstructuredReference=Zhou Q, Pang G, Zhang Z, et al. Association between gut Akkermansia and metabolic syndrome is dose-dependent and affected by microbial interactions: a cross-sectional study[J]. Diabetes Metab Syndr Obes, 2021, 14: 2177-2188., articleTitle=Association between gut Akkermansia and metabolic syndrome is dose-dependent and affected by microbial interactions: a cross-sectional study, refAbstract=null), Reference(id=1208862376116482574, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2017, volume=5, issue=1, pageStart=14, pageEnd=null, url=null, language=null, rfNumber=[19], rfOrder=18, authorNames=Li J, Zhao F, Wang Y, journalName=Microbiome, refType=null, unstructuredReference=Li J, Zhao F, Wang Y, et al. Gut microbiota dysbiosis contributes to the development of hypertension[J]. Microbiome, 2017, 5(1): 14., articleTitle=Gut microbiota dysbiosis contributes to the development of hypertension, refAbstract=null), Reference(id=1208862376221340176, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2017, volume=279, issue=1, pageStart=70, pageEnd=89, url=null, language=null, rfNumber=[20], rfOrder=19, authorNames=Pickard JM, Zeng MY, Caruso R, journalName=Immunol Rev, refType=null, unstructuredReference=Pickard JM, Zeng MY, Caruso R, et al. Gut microbiota: Role in pathogen colonization, immune responses, and inflammatory disease[J]. Immunol Rev, 2017, 279(1): 70-89., articleTitle=Gut microbiota: Role in pathogen colonization, immune responses, and inflammatory disease, refAbstract=null), Reference(id=1208862376347169307, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2018, volume=14, issue=7, pageStart=442, pageEnd=456, url=null, language=null, rfNumber=[21], rfOrder=20, authorNames=Yang T, Richards EM, Pepine CJ, journalName=Nat Rev Nephrol, refType=null, unstructuredReference=Yang T, Richards EM, Pepine CJ, et al. The gut microbiota and the brain-gut-kidney axis in hypertension and chronic kidney disease[J]. Nat Rev Nephrol, 2018, 14(7): 442-456., articleTitle=The gut microbiota and the brain-gut-kidney axis in hypertension and chronic kidney disease, refAbstract=null), Reference(id=1208862376452026910, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2015, volume=37, issue=4, pageStart=9808, pageEnd=null, url=null, language=null, rfNumber=[22], rfOrder=21, authorNames=Costello-White R, Ryff CD, Coe CL, journalName=Age (Dordr), refType=null, unstructuredReference=Costello-White R, Ryff CD, Coe CL. Aging and low-grade inflammation reduce renal function in middle-aged and older adults in Japan and the USA[J]. Age (Dordr), 2015, 37(4): 9808., articleTitle=Aging and low-grade inflammation reduce renal function in middle-aged and older adults in Japan and the USA, refAbstract=null), Reference(id=1208862376569467429, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2021, volume=9, issue=3, pageStart=225, pageEnd=null, url=null, language=null, rfNumber=[23], rfOrder=22, authorNames=Norton JE Jr, Kommineni S, Akrivoulis P, journalName=Vaccines (Basel), refType=null, unstructuredReference=Norton JE Jr, Kommineni S, Akrivoulis P, et al. Primary human dendritic cells and whole-blood based assays to evaluate immuno-modulatory properties of heat-killed commensal bacteria[J]. Vaccines (Basel), 2021, 9(3): 225., articleTitle=Primary human dendritic cells and whole-blood based assays to evaluate immuno-modulatory properties of heat-killed commensal bacteria, refAbstract=null), Reference(id=1208862376699490859, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2021, volume=12, issue=1, pageStart=e03335, pageEnd=20, url=null, language=null, rfNumber=[24], rfOrder=23, authorNames=Zhang X, Borbet TC, Fallegger A, journalName=mBio, refType=null, unstructuredReference=Zhang X, Borbet TC, Fallegger A, et al. An antibiotic-impacted microbiota compromises the development of colonic regulatory T cells and predisposes to dysregulated immune responses[J]. mBio, 2021, 12(1): e03335-20., articleTitle=An antibiotic-impacted microbiota compromises the development of colonic regulatory T cells and predisposes to dysregulated immune responses, refAbstract=null), Reference(id=1208862376795959853, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2014, volume=588, issue=22, pageStart=4188, pageEnd=4194, url=null, language=null, rfNumber=[25], rfOrder=24, authorNames=Zeissig S, Blumberg RS, journalName=FEBS Lett, refType=null, unstructuredReference=Zeissig S, Blumberg RS. Commensal microbial regulation of natural killer T cells at the frontiers of the mucosal immune system[J]. FEBS Lett, 2014, 588(22): 4188-4194., articleTitle=Commensal microbial regulation of natural killer T cells at the frontiers of the mucosal immune system, refAbstract=null), Reference(id=1208862376892428853, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2012, volume=489, issue=7415, pageStart=231, pageEnd=241, url=null, language=null, rfNumber=[26], rfOrder=25, authorNames=Maynard CL, Elson CO, Hatton RD, journalName=Nature, refType=null, unstructuredReference=Maynard CL, Elson CO, Hatton RD, et al. Reciprocal interactions of the intestinal microbiota and immune system[J]. Nature, 2012, 489(7415): 231-241., articleTitle=Reciprocal interactions of the intestinal microbiota and immune system, refAbstract=null), Reference(id=1208862376993092150, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2018, volume=54, issue=null, pageStart=385, pageEnd=400, url=null, language=null, rfNumber=[27], rfOrder=26, authorNames=Pashenkov MV, Dagil YA, Pinegin BV, journalName=Int Immunopharmacol, refType=null, unstructuredReference=Pashenkov MV, Dagil YA, Pinegin BV. NOD1 and NOD2:Molecular targets in prevention and treatment of infectious diseases[J]. Int Immunopharmacol, 2018, 54: 385-400., articleTitle=NOD1 and NOD2:Molecular targets in prevention and treatment of infectious diseases, refAbstract=null), Reference(id=1208862378180080187, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2015, volume=116, issue=6, pageStart=1022, pageEnd=1033, url=null, language=null, rfNumber=[28], rfOrder=27, authorNames=McMaster WG, Kirabo A, Madhur MS, journalName=Circ Res, refType=null, unstructuredReference=McMaster WG, Kirabo A, Madhur MS, et al. Inflammation, immunity, and hypertensive end-organ damage[J]. Circ Res, 2015, 116(6): 1022-1033., articleTitle=Inflammation, immunity, and hypertensive end-organ damage, refAbstract=null), Reference(id=1208862378276549185, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2017, volume=312, issue=3, pageStart=H368, pageEnd=H374, url=null, language=null, rfNumber=[29], rfOrder=28, authorNames=Dixon KB, Davies SS, Kirabo A, journalName=Am J Physiol Heart Circ Physiol, refType=null, unstructuredReference=Dixon KB, Davies SS, Kirabo A. Dendritic cells and isolevuglandins in immunity, inflammation, and hypertension[J]. Am J Physiol Heart Circ Physiol, 2017, 312(3): H368-H374., articleTitle=Dendritic cells and isolevuglandins in immunity, inflammation, and hypertension, refAbstract=null), Reference(id=1208862378377212486, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2017, volume=21, issue=4, pageStart=1009, pageEnd=1020, url=null, language=null, rfNumber=[30], rfOrder=29, authorNames=Barbaro NR, Foss JD, Kryshtal DO, journalName=Cell Rep, refType=null, unstructuredReference=Barbaro NR, Foss JD, Kryshtal DO, et al. Dendritic cell amiloride-sensitive channels mediate sodium-induced inflammation and hypertension[J]. Cell Rep, 2017, 21(4): 1009-1020., articleTitle=Dendritic cell amiloride-sensitive channels mediate sodium-induced inflammation and hypertension, refAbstract=null), Reference(id=1208862378490458697, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2020, volume=22, issue=10, pageStart=79, pageEnd=null, url=null, language=null, rfNumber=[31], rfOrder=30, authorNames=Elijovich F, Laffer CL, Sahinoz M, journalName=Curr Hypertens Rep, refType=null, unstructuredReference=Elijovich F, Laffer CL, Sahinoz M, et al. The gut microbiome, inflammation, and salt-sensitive hypertension[J]. Curr Hypertens Rep, 2020, 22(10): 79., articleTitle=The gut microbiome, inflammation, and salt-sensitive hypertension, refAbstract=null), Reference(id=1208862378586927691, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2019, volume=125, issue=12, pageStart=1067, pageEnd=1069, url=null, language=null, rfNumber=[32], rfOrder=31, authorNames=Pitzer AL, Kirabo A, journalName=Circ Res, refType=null, unstructuredReference=Pitzer AL, Kirabo A. Dendritic cell A20: targeting hypertension in autoimmunity[J]. Circ Res, 2019, 125(12): 1067-1069., articleTitle=Dendritic cell A20: targeting hypertension in autoimmunity, refAbstract=null), Reference(id=1208862378658230862, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2017, volume=120, issue=2, pageStart=312, pageEnd=323, url=null, language=null, rfNumber=[33], rfOrder=32, authorNames=Santisteban MM, Qi Y, Zubcevic J, journalName=Circ Res, refType=null, unstructuredReference=Santisteban MM, Qi Y, Zubcevic J, et al. Hypertension-linked pathophysiological alterations in the gut[J]. Circ Res, 2017, 120(2): 312-323., articleTitle=Hypertension-linked pathophysiological alterations in the gut, refAbstract=null), Reference(id=1208862378733728337, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2021, volume=2021, issue=null, pageStart=6633825, pageEnd=null, url=null, language=null, rfNumber=[34], rfOrder=33, authorNames=Kim JY, Lee E, Koo S, journalName=Biomed Res Int, refType=null, unstructuredReference=Kim JY, Lee E, Koo S, et al. Transfer of Th17 from adult spontaneous hypertensive rats accelerates development of hypertension in juvenile spontaneous hypertensive rats[J]. Biomed Res Int, 2021, 2021: 6633825., articleTitle=Transfer of Th17 from adult spontaneous hypertensive rats accelerates development of hypertension in juvenile spontaneous hypertensive rats, refAbstract=null), Reference(id=1208862378817614421, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2020, volume=319, issue=5, pageStart=F868, pageEnd=F875, url=null, language=null, rfNumber=[35], rfOrder=34, authorNames=La Salvia S, Musante L, Lannigan J, journalName=Am J Physiol Renal Physiol, refType=null, unstructuredReference=La Salvia S, Musante L, Lannigan J, et al. T cell-derived extracellular vesicles are elevated in essential HTN[J]. Am J Physiol Renal Physiol, 2020, 319(5): F868-F875., articleTitle=T cell-derived extracellular vesicles are elevated in essential HTN, refAbstract=null), Reference(id=1208862378914083417, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2017, volume=119, issue=null, pageStart=404, pageEnd=411, url=null, language=null, rfNumber=[36], rfOrder=35, authorNames=Rudemiller N, Crowley SD, journalName=Pharmacol Res, refType=null, unstructuredReference=Rudemiller N, Crowley SD. The role of chemokines in hypertension and consequent target organ damage[J]. Pharmacol Res, 2017, 119: 404-411., articleTitle=The role of chemokines in hypertension and consequent target organ damage, refAbstract=null), Reference(id=1208862379018941022, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2019, volume=73, issue=3, pageStart=561, pageEnd=570, url=null, language=null, rfNumber=[37], rfOrder=36, authorNames=Dingwell LS, Shikatani EA, Besla R, journalName=Hypertension, refType=null, unstructuredReference=Dingwell LS, Shikatani EA, Besla R, et al. B-cell deficiency lowers blood pressure in mice[J]. Hypertension, 2019, 73(3): 561-570., articleTitle=B-cell deficiency lowers blood pressure in mice, refAbstract=null), Reference(id=1208862379098632801, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2012, volume=7, issue=12, pageStart=1938, pageEnd=1946, url=null, language=null, rfNumber=[38], rfOrder=37, authorNames=Gupta J, Mitra N, Kanetsky PA, journalName=Clin J Am Soc Nephrol, refType=null, unstructuredReference=Gupta J, Mitra N, Kanetsky PA, et al. Association between albuminuria, kidney function, and inflammatory biomarker profile in CKD in CRIC[J]. Clin J Am Soc Nephrol, 2012, 7(12): 1938-1946., articleTitle=Association between albuminuria, kidney function, and inflammatory biomarker profile in CKD in CRIC, refAbstract=null), Reference(id=1208862379186713189, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2021, volume=22, issue=14, pageStart=7554, pageEnd=null, url=null, language=null, rfNumber=[39], rfOrder=38, authorNames=Kim H, Kim M, Lee HY, journalName=Int J Mol Sci, refType=null, unstructuredReference=Kim H, Kim M, Lee HY, et al. Role of dendritic cell in diabetic nephropathy[J]. Int J Mol Sci, 2021, 22(14): 7554., articleTitle=Role of dendritic cell in diabetic nephropathy, refAbstract=null), Reference(id=1208862379283182186, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2020, volume=98, issue=5, pageStart=1149, pageEnd=1159, url=null, language=null, rfNumber=[40], rfOrder=39, authorNames=Ponticelli C, Podestà MA, Moroni G, journalName=Kidney Int, refType=null, unstructuredReference=Ponticelli C, Podestà MA, Moroni G. Hyperuricemia as a trigger of immune response in hypertension and chronic kidney disease[J]. Kidney Int, 2020, 98(5): 1149-1159., articleTitle=Hyperuricemia as a trigger of immune response in hypertension and chronic kidney disease, refAbstract=null), Reference(id=1208862379367068269, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2016, volume=68, issue=2, pageStart=289, pageEnd=296, url=null, language=null, rfNumber=[41], rfOrder=40, authorNames=Rudemiller NP, Crowley SD, journalName=Hypertension, refType=null, unstructuredReference=Rudemiller NP, Crowley SD. Interactions between the immune and the renin-angiotensin systems in hypertension[J]. Hypertension, 2016, 68(2): 289-296., articleTitle=Interactions between the immune and the renin-angiotensin systems in hypertension, refAbstract=null), Reference(id=1208862379425788528, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2016, volume=27, issue=3, pageStart=677, pageEnd=686, url=null, language=null, rfNumber=[42], rfOrder=41, authorNames=Wenzel U, Turner JE, Krebs C, journalName=J Am Soc Nephrol, refType=null, unstructuredReference=Wenzel U, Turner JE, Krebs C, et al. Immune mechanisms in arterial hypertension[J]. J Am Soc Nephrol, 2016, 27(3): 677-686., articleTitle=Immune mechanisms in arterial hypertension, refAbstract=null), Reference(id=1208862379526451827, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2019, volume=10, issue=1, pageStart=1157, pageEnd=null, url=null, language=null, rfNumber=[43], rfOrder=42, authorNames=Cippà PE, Liu J, Sun B, journalName=Nat Commun, refType=null, unstructuredReference=Cippà PE, Liu J, Sun B, et al. A late B lymphocyte action in dysfunctional tissue repair following kidney injury and transplantation[J]. Nat Commun, 2019, 10(1): 1157., articleTitle=A late B lymphocyte action in dysfunctional tissue repair following kidney injury and transplantation, refAbstract=null), Reference(id=1208862379601949302, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2019, volume=34, issue=2, pageStart=252, pageEnd=264, url=null, language=null, rfNumber=[44], rfOrder=43, authorNames=Vila Cuenca M, van Bezu J, Beelen RHJ, journalName=Nephrol Dial Transplant, refType=null, unstructuredReference=Vila Cuenca M, van Bezu J, Beelen RHJ, et al. Stabilization of cell-cell junctions by active vitamin D ameliorates uraemia-induced loss of human endothelial barrier function[J]. Nephrol Dial Transplant, 2019, 34(2): 252-264., articleTitle=Stabilization of cell-cell junctions by active vitamin D ameliorates uraemia-induced loss of human endothelial barrier function, refAbstract=null), Reference(id=1208862379711001207, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2018, volume=10, issue=10, pageStart=404, pageEnd=null, url=null, language=null, rfNumber=[45], rfOrder=44, authorNames=Maciel RAP, Cunha RS, Busato V, journalName=Toxins (Basel), refType=null, unstructuredReference=Maciel RAP, Cunha RS, Busato V, et al. Uremia impacts VE-cadherin and ZO-1 expression in human endothelial cell-to-cell junctions[J]. Toxins (Basel), 2018, 10(10): 404., articleTitle=Uremia impacts VE-cadherin and ZO-1 expression in human endothelial cell-to-cell junctions, refAbstract=null), Reference(id=1208862379815858810, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2016, volume=7, issue=14, pageStart=17479, pageEnd=17491, url=null, language=null, rfNumber=[46], rfOrder=45, authorNames=Ding W, Guo H, Xu C, journalName=Oncotarget, refType=null, unstructuredReference=Ding W, Guo H, Xu C, et al. Mitochondrial reactive oxygen species-mediated NLRP3 inflammasome activation contributes to aldosterone-induced renal tubular cells injury[J]. Oncotarget, 2016, 7(14): 17479-17491., articleTitle=Mitochondrial reactive oxygen species-mediated NLRP3 inflammasome activation contributes to aldosterone-induced renal tubular cells injury, refAbstract=null), Reference(id=1208862379950076542, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2021, volume=117, issue=8, pageStart=1859, pageEnd=1876, url=null, language=null, rfNumber=[47], rfOrder=46, authorNames=Hall JE, Mouton AJ, da Silva AA, journalName=Cardiovasc Res, refType=null, unstructuredReference=Hall JE, Mouton AJ, da Silva AA, et al. Obesity, kidney dysfunction, and inflammation: interactions in hypertension[J]. Cardiovasc Res, 2021, 117(8): 1859-1876., articleTitle=Obesity, kidney dysfunction, and inflammation: interactions in hypertension, refAbstract=null), Reference(id=1208862380067517059, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2016, volume=167, issue=5, pageStart=1339, pageEnd=1353, url=null, language=null, rfNumber=[48], rfOrder=47, authorNames=Desai MS, Seekatz AM, Koropatkin NM, journalName=Cell, refType=null, unstructuredReference=Desai MS, Seekatz AM, Koropatkin NM, et al. A dietary fiber-deprived gut microbiota degrades the colonic mucus barrier and enhances pathogen susceptibility[J]. Cell, 2016, 167(5): 1339-1353., articleTitle=A dietary fiber-deprived gut microbiota degrades the colonic mucus barrier and enhances pathogen susceptibility, refAbstract=null), Reference(id=1208862380163986056, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2016, volume=5, issue=4, pageStart=e73, pageEnd=null, url=null, language=null, rfNumber=[49], rfOrder=48, authorNames=Corrêa-Oliveira R, Fachi JL, Vieira A, journalName=Clin Transl Immunology, refType=null, unstructuredReference=Corrêa-Oliveira R, Fachi JL, Vieira A, et al. Regulation of immune cell function by short-chain fatty acids[J]. Clin Transl Immunology, 2016, 5(4): e73., articleTitle=Regulation of immune cell function by short-chain fatty acids, refAbstract=null), Reference(id=1208862380247872140, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2015, volume=26, issue=8, pageStart=1877, pageEnd=1888, url=null, language=null, rfNumber=[50], rfOrder=49, authorNames=Andrade-Oliveira V, Amano MT, Correacosta M, journalName=J Am Soc Nephrol, refType=null, unstructuredReference=Andrade-Oliveira V, Amano MT, Correacosta M, et al. Gut bacteria products prevent AKI induced by ischemia-reperfusion[J]. J Am Soc Nephrol, 2015, 26(8): 1877-1888., articleTitle=Gut bacteria products prevent AKI induced by ischemia-reperfusion, refAbstract=null), Reference(id=1208862380340146834, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2018, volume=132, issue=6, pageStart=701, pageEnd=718, url=null, language=null, rfNumber=[51], rfOrder=50, authorNames=Kim S, Goel R, Kumar A, journalName=Clin Sci (Lond), refType=null, unstructuredReference=Kim S, Goel R, Kumar A, et al. Imbalance of gut microbiome and intestinal epithelial barrier dysfunction in patients with high blood pressure[J]. Clin Sci (Lond), 2018, 132(6): 701-718., articleTitle=Imbalance of gut microbiome and intestinal epithelial barrier dysfunction in patients with high blood pressure, refAbstract=null), Reference(id=1208862380461781652, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2018, volume=11, issue=1, pageStart=51, pageEnd=null, url=null, language=null, rfNumber=[52], rfOrder=51, authorNames=de la Cuesta-Zuluaga J, Mueller NT, Álvarez-Quintero R, journalName=Nutrients, refType=null, unstructuredReference=de la Cuesta-Zuluaga J, Mueller NT, Álvarez-Quintero R, et al. Higher fecal short-chain fatty acid levels are associated with gut microbiome dysbiosis, obesity, hypertension and cardiometabolic disease risk factors[J]. Nutrients, 2018, 11(1): 51., articleTitle=Higher fecal short-chain fatty acid levels are associated with gut microbiome dysbiosis, obesity, hypertension and cardiometabolic disease risk factors, refAbstract=null), Reference(id=1208862380575027864, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2019, volume=74, issue=1, pageStart=184, pageEnd=193, url=null, language=null, rfNumber=[53], rfOrder=52, authorNames=Cheema MU, Pluznick JL, journalName=Hypertension, refType=null, unstructuredReference=Cheema MU, Pluznick JL. Gut microbiota plays a central role to modulate the plasma and fecal metabolomes in response to angiotensin II[J]. Hypertension, 2019, 74(1): 184-193., articleTitle=Gut microbiota plays a central role to modulate the plasma and fecal metabolomes in response to angiotensin II, refAbstract=null), Reference(id=1208862380684079774, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2017, volume=91, issue=6, pageStart=1336, pageEnd=1346, url=null, language=null, rfNumber=[54], rfOrder=53, authorNames=Wysocki J, Ye M, Khattab AM, journalName=Kidney International, refType=null, unstructuredReference=Wysocki J, Ye M, Khattab AM, et al. Angiotensin-converting enzyme 2 amplification limited to the circulation does not protect mice from development of diabetic nephropathy[J]. Kidney International, 2017, 91(6): 1336-1346., articleTitle=Angiotensin-converting enzyme 2 amplification limited to the circulation does not protect mice from development of diabetic nephropathy, refAbstract=null), Reference(id=1208862380763771553, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2018, volume=69, issue=null, pageStart=167, pageEnd=179, url=null, language=null, rfNumber=[55], rfOrder=54, authorNames=Yisireyili M, Uchida Y, Yamamoto K, journalName=Brain Behav Immun, refType=null, unstructuredReference=Yisireyili M, Uchida Y, Yamamoto K, et al. Angi otensin receptor blocker irbesartan reduces stress-induced intestinal inflammation via AT1a signaling and ACE2-dependent mechanism in mice[J]. Brain Behav Immun, 2018, 69: 167-179., articleTitle=Angi otensin receptor blocker irbesartan reduces stress-induced intestinal inflammation via AT1a signaling and ACE2-dependent mechanism in mice, refAbstract=null), Reference(id=1208862380826686117, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2013, volume=167, issue=2, pageStart=131, pageEnd=137, url=null, language=null, rfNumber=[56], rfOrder=55, authorNames=Gonzalez-Gonzalez C, Gibson T, Jauregi P, journalName=Int J Food Microbiol, refType=null, unstructuredReference=Gonzalez-Gonzalez C, Gibson T, Jauregi P. Novel probiotic-fermented milk with angiotensin I-converting enzyme inhibitory peptides produced by Bifidobacterium bifidum MF 20/5[J]. Int J Food Microbiol, 2013, 167(2): 131-137., articleTitle=Novel probiotic-fermented milk with angiotensin I-converting enzyme inhibitory peptides produced by Bifidobacterium bifidum MF 20/5, refAbstract=null), Reference(id=1208862380906377897, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2016, volume=76, issue=null, pageStart=30, pageEnd=44, url=null, language=null, rfNumber=[57], rfOrder=56, authorNames=Dave LA, Hayes M, Montoya CA, journalName=Peptides, refType=null, unstructuredReference=Dave LA, Hayes M, Montoya CA, et al. Human gut endogenous proteins as a potential source of angiotensin-I-converting enzyme (ACE-I)-, renin inhibitory and antioxidant peptides[J]. Peptides, 2016, 76: 30-44., articleTitle=Human gut endogenous proteins as a potential source of angiotensin-I-converting enzyme (ACE-I)-, renin inhibitory and antioxidant peptides, refAbstract=null), Reference(id=1208862381015429804, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2015, volume=47, issue=10, pageStart=443, pageEnd=446, url=null, language=null, rfNumber=[58], rfOrder=57, authorNames=Honour JW, journalName=Physiol Genomics, refType=null, unstructuredReference=Honour JW. Historical perspective: gut dysbiosis and hypertension[J]. Physiol Genomics, 2015, 47(10): 443-446., articleTitle=Historical perspective: gut dysbiosis and hypertension, refAbstract=null), Reference(id=1208862381128676018, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2014, volume=30, issue=12, pageStart=1700, pageEnd=1705, url=null, language=null, rfNumber=[59], rfOrder=58, authorNames=Ufnal M, Jazwiec R, Dadlez M, journalName=Can J Cardiol, refType=null, unstructuredReference=Ufnal M, Jazwiec R, Dadlez M, et al. Trimethylamine-N-oxide:a carnitine-derived metabolite that prolongs the hypertensive effect of angiotensin Ⅱ in rats[J]. Can J Cardiol, 2014, 30(12): 1700-1705., articleTitle=Trimethylamine-N-oxide:a carnitine-derived metabolite that prolongs the hypertensive effect of angiotensin Ⅱ in rats, refAbstract=null), Reference(id=1208862381237727927, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2021, volume=134, issue=null, pageStart=111156, pageEnd=null, url=null, language=null, rfNumber=[60], rfOrder=59, authorNames=Naqvi S, Asar TO, Kumar V, journalName=Biomed Pharmacother, refType=null, unstructuredReference=Naqvi S, Asar TO, Kumar V, et al. A cross-talk between gut microbiome, salt and hypertension[J]. Biomed Pharmacother, 2021, 134: 111156., articleTitle=A cross-talk between gut microbiome, salt and hypertension, refAbstract=null), Reference(id=1208862381330002617, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2020, volume=11, issue=1, pageStart=66, pageEnd=76, url=null, language=null, rfNumber=[61], rfOrder=60, authorNames=Ge X, Zheng L, Zhuang R, journalName=Adv Nutr, refType=null, unstructuredReference=Ge X, Zheng L, Zhuang R, et al. The gut microbial metabolite trimethylamine N-oxide and hypertension risk: a systematic review and dose-response meta-analysis[J]. Adv Nutr, 2020, 11(1): 66-76., articleTitle=The gut microbial metabolite trimethylamine N-oxide and hypertension risk: a systematic review and dose-response meta-analysis, refAbstract=null), Reference(id=1208862381430665916, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2019, volume=9, issue=1, pageStart=4249, pageEnd=null, url=null, language=null, rfNumber=[62], rfOrder=61, authorNames=Chou RH, Chen CY, Chen IC, journalName=Sci Rep, refType=null, unstructuredReference=Chou RH, Chen CY, Chen IC, et al. Trimethylamine N-oxide, circulating endothelial progenitor cells, and endothelial function in patients with stable angina[J]. Sci Rep, 2019, 9(1): 4249., articleTitle=Trimethylamine N-oxide, circulating endothelial progenitor cells, and endothelial function in patients with stable angina, refAbstract=null), Reference(id=1208862382659596992, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2017, volume=8, issue=null, pageStart=139, pageEnd=null, url=null, language=null, rfNumber=[63], rfOrder=62, authorNames=Chen K, Zheng X, Feng M, journalName=Front Physiol, refType=null, unstructuredReference=Chen K, Zheng X, Feng M, et al. Gut microbiota-dependent metabolite trimethylamine N-oxide contributes to cardiac dysfunction in western diet-induced obese mice[J]. Front Physiol, 2017, 8: 139., articleTitle=Gut microbiota-dependent metabolite trimethylamine N-oxide contributes to cardiac dysfunction in western diet-induced obese mice, refAbstract=null), Reference(id=1208862382756065988, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2016, volume=146, issue=2, pageStart=283, pageEnd=289, url=null, language=null, rfNumber=[64], rfOrder=63, authorNames=Eckardstein A, Müller D, journalName=J Nutr, refType=null, unstructuredReference=Eckardstein A, Müller D. Plasma concentrations of trimethylamine-N-oxide are directly associated with dairy food consumption and low-grade inflammation in a German adult population[J]. J Nutr, 2016, 146(2): 283-289., articleTitle=Plasma concentrations of trimethylamine-N-oxide are directly associated with dairy food consumption and low-grade inflammation in a German adult population, refAbstract=null), Reference(id=1208862382856729286, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2016, volume=481, issue=1-2, pageStart=63, pageEnd=70, url=null, language=null, rfNumber=[65], rfOrder=64, authorNames=Sun X, Jiao X, Ma Y, journalName=Biochem Biophys Res Commun, refType=null, unstructuredReference=Sun X, Jiao X, Ma Y, et al. Trimethylamine N-oxide induces inflammation and endothelial dysfunction in human umbilical vein endothelial cells via activating ROS-TXNIP-NLRP3 inflammasome[J]. Biochem Biophys Res Commun, 2016, 481(1-2): 63-70., articleTitle=Trimethylamine N-oxide induces inflammation and endothelial dysfunction in human umbilical vein endothelial cells via activating ROS-TXNIP-NLRP3 inflammasome, refAbstract=null), Reference(id=1208862382944809673, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2017, volume=37, issue=2, pageStart=BSR20160244, pageEnd=null, url=null, language=null, rfNumber=[66], rfOrder=65, authorNames=Ma G, Pan B, Chen Y, journalName=Biosci Rep, refType=null, unstructuredReference=Ma G, Pan B, Chen Y, et al. Trimethylamine N-oxide in atherogenesis: impairing endothelial self-repair capacity and enhancing monocyte adhesion[J]. Biosci Rep, 2017, 37(2): BSR20160244., articleTitle=Trimethylamine N-oxide in atherogenesis: impairing endothelial self-repair capacity and enhancing monocyte adhesion, refAbstract=null), Reference(id=1208862383070638797, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2016, volume=5, issue=2, pageStart=e002767, pageEnd=null, url=null, language=null, rfNumber=[67], rfOrder=66, authorNames=Seldin MM, Meng Y, Qi H, journalName=J Am Heart Assoc, refType=null, unstructuredReference=Seldin MM, Meng Y, Qi H, et al. Trimethylamine N-oxide promotes vascular inflammation through signaling of mitogen-activated protein kinase and nuclear factor-κB[J]. J Am Heart Assoc, 2016, 5(2): e002767., articleTitle=Trimethylamine N-oxide promotes vascular inflammation through signaling of mitogen-activated protein kinase and nuclear factor-κB, refAbstract=null), Reference(id=1208862383179690704, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1208862369971826940, doi=null, pmid=null, pmcid=null, year=2017, volume=493, issue=2, pageStart=964, pageEnd=970, url=null, language=null, rfNumber=[68], rfOrder=67, authorNames=Sun G, Yin Z, Liu N, journalName=Biochem Biophys Res Commun, refType=null, unstructuredReference=Sun G, Yin Z, Liu N, et al. Gut microbial metabolite TMAO contributes to renal dysfunction in a mouse model of diet-induced obesity[J]. 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