Article(id=1207433497950655396, tenantId=1146029695717560320, journalId=1189873630562394117, issueId=1207433493215289544, articleNumber=null, orderNo=null, doi=10.11855/j.issn.0577-7402.2022.08.0831, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=research-article, receivedDate=1624723200000, receivedDateStr=2021-06-27, revisedDate=null, revisedDateStr=null, acceptedDate=1628524800000, acceptedDateStr=2021-08-10, onlineDate=1765804178941, onlineDateStr=2025-12-15, pubDate=1661616000000, pubDateStr=2022-08-28, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1765804178941, onlineIssueDateStr=2025-12-15, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1765804178941, creator=13701087609, updateTime=1765804178941, updator=13701087609, issue=Issue{id=1207433493215289544, tenantId=1146029695717560320, journalId=1189873630562394117, year='2022', volume='47', issue='8', pageStart='745', pageEnd='850', issueExtLink='null', onlineDate='null', pubDate='null', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=0, createTime=1765804177811, creator=13701087609, updateTime=1765804292764, updator=13701087609, preIssue=null, nextIssue=null, ext={EN=IssueExt(id=1207433975413444883, tenantId=1146029695717560320, journalId=1189873630562394117, issueId=1207433493215289544, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1207433975413444884, tenantId=1146029695717560320, journalId=1189873630562394117, issueId=1207433493215289544, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null}, startPage=831, endPage=837, ext={EN=ArticleExt(id=1207433498286199734, articleId=1207433497950655396, tenantId=1146029695717560320, journalId=1189873630562394117, language=EN, title=Research progress of Wnt/β-catenin signaling pathway in cancer stem cells, columnId=1190243275882729994, journalTitle=Medical Journal of Chinese People’s Liberation Army, columnName=Review, runingTitle=null, highlight=null, articleAbstract=

The incidence and mortality of malignant tumors are gradually increasing all the world, and the existing diagnosis and treatment methods still can not effectively block the occurrence and development of tumors. Some studies have found that cancer stem cells (CSCs) are the cells of tumor origin, and closely related to the occurrence, development and metastasis of tumors.Early detection and intervention with effective treatment of CSCs are expected to prevent the occurrence and development of tumors. Many studies have shown that the activation of Wnt/β-catenin signaling pathway is an important link in the initiation of CSCs and the formation of tumors. Therefore, by regulating the key upstream and downstream factors of the Wnt/β-catenin pathway in CSCs, thereby regulating the activity of the Wnt/β-catenin signaling pathway, controlling tumor growth and metastasis,can provide new targets and directions for the treatment and prevention of malignant tumors. The key role of Wnt/β-catenin signal pathway in CSCs, and the related mechanisms of the important factors of the pathway in regulating the tumor growth, development and metastasis of CSCs in different organs are reviewed in present paper.

, correspAuthors=Hui Zhao, authorNote=null, correspAuthorsNote=, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, authorCompany=null, fund=null, authors=null, authorsList=Yu-Jia Tian, Ming Wei, Hui Zhao), CN=ArticleExt(id=1207433498768544715, articleId=1207433497950655396, tenantId=1146029695717560320, journalId=1189873630562394117, language=CN, title=Wnt/β-catenin信号通路在肿瘤干细胞中的作用研究进展, columnId=1190243276029530637, journalTitle=解放军医学杂志, columnName=综述, runingTitle=null, highlight=null, articleAbstract=

全球恶性肿瘤的发病率及死亡率呈逐渐升高趋势，现有的诊疗手段仍不能有效阻断肿瘤的发生和发展。肿瘤干细胞(CSCs)是肿瘤的起源细胞，与肿瘤的发生、发展及转移密切相关。若早期发现CSCs并有效的治疗方式进行干预，有望阻止肿瘤的发生与发展。Wnt/β-catenin信号通路的激活是CSCs启动及肿瘤形成的重要环节，通过调节CSCs中Wnt/β-catenin信号通路的上下游关键因子来调控该通路的活性，控制肿瘤的生长及转移，可为恶性肿瘤的治疗与预防提供新的靶点和方向。本文对Wnt/β-catenin信号通路在CSCs中的作用，以及该通路中关键因子调控CSCs在不同脏器肿瘤发生、发展及转移中的相关机制进行综述。

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