Article(id=1199335055475700519, tenantId=1146029695717560320, journalId=1189873630562394117, issueId=1199335049175859209, articleNumber=null, orderNo=null, doi=10.11855/j.issn.0577-7402.0972.2023.1020, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=research-article, receivedDate=1689696000000, receivedDateStr=2023-07-19, revisedDate=null, revisedDateStr=null, acceptedDate=1693756800000, acceptedDateStr=2023-09-04, onlineDate=1763873359792, onlineDateStr=2025-11-23, pubDate=1711555200000, pubDateStr=2024-03-28, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1763873359792, onlineIssueDateStr=2025-11-23, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1763873359792, creator=13701087609, updateTime=1763873359792, updator=13701087609, issue=Issue{id=1199335049175859209, tenantId=1146029695717560320, journalId=1189873630562394117, year='2024', volume='49', issue='3', pageStart='245', pageEnd='366', issueExtLink='null', onlineDate='null', pubDate='null', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=-1, specialIssue=0, createTime=1763873358291, creator=13701087609, updateTime=1763874044185, updator=13701087609, preIssue=null, nextIssue=null, ext={EN=IssueExt(id=1199337926086721596, tenantId=1146029695717560320, journalId=1189873630562394117, issueId=1199335049175859209, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1199337926086721597, tenantId=1146029695717560320, journalId=1189873630562394117, issueId=1199335049175859209, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null}, startPage=355, endPage=359, ext={EN=ArticleExt(id=1199335056935318353, articleId=1199335055475700519, tenantId=1146029695717560320, journalId=1189873630562394117, language=EN, title=Research progress on the clinical classification correlation between liver cirrhosis and liver failure, columnId=1190243275882729994, journalTitle=Medical Journal of Chinese People’s Liberation Army, columnName=Review, runingTitle=null, highlight=null, articleAbstract=
The essence of cirrhosis is the over-repairing reaction of liver tissue damage in the process of chronic liver disease. During repair, the liver parenchyma is gradually replaced by fibrosis tissue, resulting in changes in liver tissue morphology, followed by portal hypertension and other related manifestations. Liver failure are serious disorder of liver functions (synthesis, metabolism, transformation, regeneration, etc.) caused by various factors, often mainly manifested as jaundice, coagulation disfunction, hepatic encephalopathy, ascites, etc. The naming and typing of the two are different, and they can exist independently of each other or intersect with each other. In recent years, with the in-depth exploration of cirrhosis and liver failure, many new definitions and classification methods have been put forward in the research. However, due to the confusion of classification methods, there is still a lack of summary, so this article briefly reviews the current progress of clinical classification of liver cirrhosis and liver failure and their differences and intersections.
, correspAuthors=Shan-Hong Tang, authorNote=null, correspAuthorsNote=
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肝硬化本质是慢性肝病过程中肝组织损伤的过度修复反应,修复时肝实质逐渐被纤维化组织取代,导致肝脏组织形态学发生改变,继而出现门静脉高压症等相关表现。肝衰竭是由各种因素引起的肝脏多种功能(合成、代谢、生物转化、再生等)发生严重障碍,常以黄疸、凝血功能障碍、肝性脑病、腹水等为主要表现。二者的命名及分型的关注点有差别,相互之间可独立存在,也可彼此交集。近年来随着对肝硬化及肝衰竭的深入探索,提出了许多新的定义及分类方法,但分类方法混杂,缺乏有效归纳总结。本文就目前肝硬化与肝衰竭临床分类的进展,以及二者的差异与交集进行综述。
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1Department of Gastroenterology, the General Hospital of Western Theater Command, Chengdu, Sichuan 610083, China
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1解放军西部战区总医院消化内科,四川成都 610083
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1Department of Gastroenterology, the General Hospital of Western Theater Command, Chengdu, Sichuan 610083, China
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1解放军西部战区总医院消化内科,四川成都 610083
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1解放军西部战区总医院消化内科,四川成都 610083
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75(Suppl 1): S49-S66., articleTitle=Pathophysiology of decompensated cirrhosis: portal hypertension, circulatory dysfunction, inflammation, metabolism and mitochondrial dysfunction, refAbstract=null)], funds=null, companyList=[AuthorCompany(id=1199335058160055191, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1199335055475700519, xref=1, ext=[AuthorCompanyExt(id=1199335058168443801, tenantId=1146029695717560320, journalId=1189873630562394117, articleId=1199335055475700519, companyId=1199335058160055191, language=EN, country=null, province=null, city=null, postcode=null, companyName=null, departmentName=null, remark=
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肝硬化疾病进展过程与肝衰竭的关系ACLF. 慢加急性肝衰竭;SDC. 稳定性失代偿;UDC. 不稳定性失代偿;Pre-ACLF. 慢加急性肝衰竭前期;AD. 急性失代偿;NAD. 非急性失代偿
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