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Lung cancer is one of the most common cancers worldwide. As reported in 2020, there were approximately 2.2 million new cases of lung cancer worldwide, accounting for 11.4% of newly diagnosed malignant tumors. Additionally, there were 1.8 million deaths resulting from lung cancer in 2020, representing about 18% of all cancer-related deaths globally. Lung cancer remains the leading cause of cancer-associated morbidity and mortality in China. Despite the increasing popularity of immunotherapy and targeted therapy, chemotherapy remains one of the most dominant treatment modalities for lung cancer (particularly for small-cell lung cancer). However, chemotherapeutic drugs in clinical practice lack specificity and kill normal cells while killing tumor cells, especially cells that are relatively active in proliferation or metabolism. Chemotherapy-induced myelosuppression (CIM) is the most common dose-limiting and potentially fatal complication in cancer therapy and a key factor affecting the course and dose of chemotherapy, and nearly 90% of chemotherapeutic drugs may be associated with myelosuppression. Myelosuppression is the biggest obstacle to cancer chemotherapy. Myelosuppression seriously affects the life quality of cancer patients, reduces patient compliance, delays treatment, and even threatens the lives of patients, which we should pay attention to in clinical practice. At present, the main treatment regimens for bone marrow suppression are those follows, such as drugs promoting neutrophils, red blood cells, thrombopoietic drugs and blood transfusion. As the world's first approved CDK4/6 inhibitor with full myeloprotection, the use of trilaciclib before chemotherapy is effective in reducing the incidences of CIM. This review aims to summarize the relevant research progress in recent years from the mechanism of bone marrow suppression, related treatment measures of bone marrow suppression after chemotherapy for lung cancer, and provides further ideas and clinical basis for clinical application, monitoring and management of CIM.

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肺癌是全球最常见的癌症之一。2020年研究数据表明，全球新发肺癌病例约220万，占所有新发恶性肿瘤的11.4%。2020年，全球肺癌死亡人数约180万，约占恶性肿瘤相关死亡的18%。在中国，肺癌仍是发病率和死亡率第一的癌症。尽管免疫治疗及靶向治疗日益普及，化疗目前仍然是肺癌（特别是小细胞肺癌）最主要的治疗方式之一。然而，目前临床常用的化疗药物缺乏特异性，在杀死肿瘤细胞的同时也会杀伤正常细胞，特别是对增殖或代谢比较活跃的细胞。化疗引起的骨髓抑制是最常见的化疗非特异性毒性，也是影响化疗疗程及剂量的关键因素，接近90%的化疗药物可出现骨髓抑制情况。骨髓抑制是肿瘤化疗的最大障碍，骨髓抑制严重影响了肿瘤患者的生活质量，降低患者的依从性，延迟治疗，甚至会威胁患者的生命，应得到临床充分的重视。目前，针对化疗后骨髓抑制的治疗方法主要有促进中性粒细胞、红细胞、血小板生成药物及输血等。作为全球首个获批的具有全系骨髓保护作用的细胞周期蛋白依赖性激酶（CDK）4/6抑制剂，化疗前使用曲拉西利可从源头上有效降低化疗引起的骨髓抑制发生率。本综述拟通过探讨骨髓抑制相关机制、肺癌化疗后骨髓抑制的相关治疗措施等总结近年来的相关研究进展，为后续临床应用、监测和管理化疗相关的骨髓抑制提供进一步的思路和临床依据。

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吴育锋，男，副主任医师，主要从事小细胞肺癌的诊断和治疗。E-mail: yufengwu@vip.qq.com。

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吴育锋，男，副主任医师，主要从事小细胞肺癌的诊断和治疗。E-mail: yufengwu@vip.qq.com。

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