Article(id=1321826809914708857, tenantId=1146029695717560320, journalId=1227999776411738115, issueId=1321826809193288569, articleNumber=null, orderNo=null, doi=10.12048/j.issn.1674-229X.2026.07.002, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=1762272000000, receivedDateStr=2025-11-05, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1793077669837, onlineDateStr=2026-10-27, pubDate=1784908800000, pubDateStr=2026-07-25, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1793077669837, onlineIssueDateStr=2026-10-27, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1793077669837, creator=13701087609, updateTime=1793077669837, updator=13701087609, issue=Issue{id=1321826809193288569, tenantId=1146029695717560320, journalId=1227999776411738115, year='2026', volume='36', issue='7', pageStart='481', pageEnd='560', issueExtLink='null', onlineDate='null', pubDate='1784908800000', pubDateStr='2026-07-25', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=1, specialIssue=null, createTime=1793077669666, creator='13701087609', updateTime=1793077915753, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1321827841440858287, tenantId=1146029695717560320, journalId=1227999776411738115, issueId=1321826809193288569, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1321827841440858288, tenantId=1146029695717560320, journalId=1227999776411738115, issueId=1321826809193288569, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=492, endPage=497, ext={EN=ArticleExt(id=1321826810107646842, articleId=1321826809914708857, tenantId=1146029695717560320, journalId=1227999776411738115, language=EN, title=Development of A Functional Food with Homology between Medicine and Food for Auxiliary Protection of Gastric Mucosal Injury, columnId=null, journalTitle=Pharmacy Today, columnName=null, runingTitle=null, highlight=null, articleAbstract=
OBJECTIVE Based on the theory of traditional Chinese medicine，to develop a functional food with homology between medicine and food，composed of Hericium erinaceus，Astragalus radix，Paeonia radix alba，Citri reticulatae pericarpium，Amomi fructus，Magrarita pulveratum，etc.，and to evaluate its auxiliary protective function against gastric mucosal injury.
METHODS The anti-Helicobacter pylori activity was evaluated through in vitro experiments. The rat model of alcohol-induced gastric mucosal injury was adopted to evaluate its protective effect on gastric mucosa. The health care function of assisting in protecting gastric mucosa was evaluated through human clinical trials.
RESULTS In vitro experiments indicated that this functional food had inhibitory and killing effects on the standard strain of Helicobacter pylori (ATCC 700392)，with a minimum inhibitory concentration (MIC) of 75 mg·mL-1 and a minimum bactericidal concentration (MBC) of 150 mg·mL-1. Animal experiments showed that the total lesion score of the gastric mucosa histopathological examination in the 4.50 g·kg-1 BW dose group was significantly lower than that in the model control group (P<0.05). The gastric mucosal injury score indices in the 1.50 g·kg-1 BW and 4.50 g·kg-1 BW dose groups were also significantly lower than those in the model control group，indicating an auxiliary protective effect on mucosal injury. Human clinical trials revealed that，compared with their own baseline before the trial，the clinical symptom and sign scores related to gastric mucosal injury in the trial group were significantly reduced (P<0.05).
CONCLUSION This study successfully develops a functional food with a clear auxiliary protective function against gastric mucosal injury. Its mechanism of action may be related to the inhibition of Helicobacter pylori and the enhancement of gastric mucosal defense，and it has a good application prospect.
, authors=Zongsheng YU
1, 2, Yuexin TIAN
1, Yulan DU
1, authorsList=Zongsheng YU, Yuexin TIAN, Yulan DU, authorCompany=null, correspAuthors=null, authorNote=null, correspAuthorsNote=null, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1321826810464162683, articleId=1321826809914708857, tenantId=1146029695717560320, journalId=1227999776411738115, language=CN, title=一种对胃黏膜损伤有辅助保护功能的药食同源功能性食品的开发, columnId=1228268589086606089, journalTitle=今日药学, columnName=论著, runingTitle=null, highlight=null, articleAbstract=
目的 基于中医药理论，开发一种由猴头菇、黄芪、白芍、陈皮、砂仁、珍珠粉等组成的药食同源功能性食品，并评价其对胃黏膜损伤的辅助保护功能。
方法 通过体外实验评价其抗幽门螺杆菌活性；采用大鼠酒精性胃黏膜损伤模型，评价其胃黏膜保护作用；通过人体临床试验评价辅助保护胃黏膜的保健功能。
结果 体外实验表明，该功能性食品对幽门螺杆菌标准菌株（ATCC 700392）具有抑制和杀灭作用，最小抑菌浓度（MIC）为75 mg·mL-1，最小杀菌浓度（MBC）为150 mg·mL-1。动物实验显示，4.50 g·kg-1 BW剂量组大鼠的胃黏膜病理组织学总积分较模型对照组显著下降（P<0.05）；1.50 g·kg-1 BW与4.50 g·kg-1 BW组大鼠的胃黏膜损伤积分指数亦均显著低于模型对照组，提示其具备辅助保护作用。人群试食结果显示，试食组在服用产品后与自身基线相比，胃黏膜损伤相关的临床症状及体征积分均显著降低（P<0.05）。
结论 本研究成功开发出一种对胃黏膜损伤具有明确辅助保护功能的功能性食品，其作用机制可能与抑制幽门螺杆菌、增强胃黏膜防御有关，具备良好的应用前景。
, authors=余宗盛
1, 2, 田悦昕
1, 杜玉兰
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