Article(id=1292765532143513780, tenantId=1146029695717560320, journalId=1291416979304972313, issueId=1291824268032110598, articleNumber=null, orderNo=null, doi=10.3969/j.issn.1005-4847.2026.03.011, pmid=null, cstr=null, oa=null, hot=null, price=null, onlineType=0, articleFormat=0, articleType=null, articleTypeStr=null, receivedDate=1750003200000, receivedDateStr=2025-06-16, revisedDate=null, revisedDateStr=null, acceptedDate=null, acceptedDateStr=null, onlineDate=1786148921284, onlineDateStr=2026-08-08, pubDate=1774800000000, pubDateStr=2026-03-30, doiRegisterDate=null, doiRegisterDateStr=null, onlineIssueDate=1786148921284, onlineIssueDateStr=2026-08-08, onlineJustAcceptDate=null, onlineJustAcceptDateStr=null, onlineFirstDate=null, onlineFirstDateStr=null, sourceXml=null, magXml=null, createTime=1786148921284, creator=13701087609, updateTime=1786148921284, updator=13701087609, issue=Issue{id=1291824268032110598, tenantId=1146029695717560320, journalId=1291416979304972313, year='2026', volume='34', issue='3', pageStart='313', pageEnd='468', issueExtLink='null', onlineDate='null', pubDate='1774800000000', pubDateStr='2026-03-30', beforeIssueId=null, nextIssueId=null, price=null, status=1, issueComplete=1, articleOrder=1, issueType=1, specialIssue=null, createTime=1785924506432, creator='ztmeta', updateTime=1786331936953, updator='13701087609', preIssue=null, nextIssue=null, articleTotal=null, ext={EN=IssueExt(id=1293533155567366441, tenantId=1146029695717560320, journalId=1291416979304972313, issueId=1291824268032110598, language=EN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=), CN=IssueExt(id=1293533155567366442, tenantId=1146029695717560320, journalId=1291416979304972313, issueId=1291824268032110598, language=CN, specialIssueTitle=, coverIllustrator=null, specialIssueEditor=, specialIssueAbout=)}, issueFiles=null, downloadFileDto=null}, startPage=432, endPage=441, ext={EN=ArticleExt(id=1292765533724766389, articleId=1292765532143513780, tenantId=1146029695717560320, journalId=1291416979304972313, language=EN, title=Research progress on regulation of the prostate cancer immune microenvironment by monoamine oxidase A and B, columnId=null, journalTitle=Acta Laboratorium Animalis Scientia Sinica, columnName=null, runingTitle=null, highlight=null, articleAbstract=

Cancer-associated fibroblasts (CAFs) influence the progression and treatment of prostate cancer (PCa) by remodeling the tumor microenvironment (TME). Identifying the key molecular drivers of this remodeling is critical for advancing cancer research. The monoamine oxidase (MAO) family, comprising the subtypes MAOA and MAOB, is a group of mitochondrial outer membrane oxidases that regulate monoamine metabolism through oxidative deamination. Aberrant MAO expression is closely associated with PCa growth, invasion, metastasis, poor prognosis, and TME remodeling. This review, based on various animal models, systematically summarizes the biological roles of the distinct MAO subtypes in CAFs and their contributions to PCa pathogenesis. We further explore the potential of MAO inhibitors in combination therapies for PCa, focusing on their differential expression profiles and functional specificity. In addition, we propose precision diagnostic and therapeutic strategies targeting MAO subtypes, with the aim of identifying novel therapeutic targets for personalized PCa treatment.

, authors=Liting FANG^{1, 2}, Yaohua HU², Fang WANG³, Jumei ZHAO^{1, *}, Changhong SHI^{2, *}, authorsList=Liting FANG, Yaohua HU, Fang WANG, Jumei ZHAO, Changhong SHI, authorCompany=null, correspAuthors=Jumei ZHAO, Changhong SHI, authorNote=null, correspAuthorsNote=, copyrightStatement=null, copyrightOwner=null, extLink=null, articleAbsUrl=null, sourceXml=null, magXml=null, pdfUrl=null, pdf=null, pdfFileSize=null, pdfExtLink=null, richHtmlUrl=null, mobilePdfUrl=null, reviewReport=null, pdfFirstPage=null, abstractGraph=null, abstractGraphContent=null, abstractVideo=null, citation=null, cebUrl=null, magXmlContent=null, mapNumber=null, fund=null), CN=ArticleExt(id=1292765534022561974, articleId=1292765532143513780, tenantId=1146029695717560320, journalId=1291416979304972313, language=CN, title=MAOA与MAOB调控前列腺癌免疫微环境的研究进展, columnId=1292765517996122928, journalTitle=中国实验动物学报, columnName=研究进展, runingTitle=null, highlight=null, articleAbstract=

癌相关成纤维细胞（cancer-associated fibroblasts, CAFs）通过重塑肿瘤微环境（tumor microenvironment, TME）显著影响前列腺癌（prostate cancer, PCa）的发展和治疗，解析驱动其重塑的关键分子对于肿瘤研究具有重要意义。单胺氧化酶（monoamine oxidase, MAO）家族是具有两种亚型MAOA和MAOB的线粒体外膜氧化酶，通过氧化脱氨作用参与单胺类物质代谢，其异常表达与PCa的生长、侵袭、迁移及不良预后密切相关，并显著影响TME的重塑。本文基于不同类型的动物模型，系统综述不同MAO亚型在CAFs中的生物学特性及其在PCa中的作用，探讨MAO抑制剂（monoamine oxidase inhibitor, MAOI）联合靶向治疗PCa的潜在价值，重点关注其在PCa中差异表达的特征，进一步提出了开发基于不同MAO亚型的精准诊疗策略，期望为PCa的个体化治疗提供有效的干预靶点。

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房丽婷，女，在读硕士研究生，研究方向：肿瘤免疫。Email：15891108115@163.com

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房丽婷，女，在读硕士研究生，研究方向：肿瘤免疫。Email：15891108115@163.com

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